• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Clinical response to crizotinib retreatment after acquisition of drug resistance.

作者信息

Matsuoka Hiromichi, Kurata Takayasu, Okamoto Isamu, Kaneda Hiroyasu, Tanaka Kaoru, Nakagawa Kazuhiko

机构信息

Department of Medical Oncology, Kinki University Faculty of Medicine, Osaka-Sayama, Osaka, Japan.

出版信息

J Clin Oncol. 2013 Jul 1;31(19):e322-3. doi: 10.1200/JCO.2012.46.4305. Epub 2013 May 28.

DOI:10.1200/JCO.2012.46.4305
PMID:23715571
Abstract
摘要

相似文献

1
Clinical response to crizotinib retreatment after acquisition of drug resistance.获得耐药性后对克唑替尼再治疗的临床反应。
J Clin Oncol. 2013 Jul 1;31(19):e322-3. doi: 10.1200/JCO.2012.46.4305. Epub 2013 May 28.
2
New insights into anaplastic lymphoma kinase-positive nonsmall cell lung cancer.间变性淋巴瘤激酶阳性非小细胞肺癌的新见解
Indian J Cancer. 2017 Jan-Mar;54(1):203-208. doi: 10.4103/ijc.IJC_72_17.
3
Long-term response to gefitinib and crizotinib in lung adenocarcinoma harboring both epidermal growth factor receptor mutation and EML4-ALK fusion gene.表皮生长因子受体突变和EML4-ALK融合基因均阳性的肺腺癌患者对吉非替尼和克唑替尼的长期反应
J Clin Oncol. 2014 Mar 20;32(9):e30-2. doi: 10.1200/JCO.2012.47.7141. Epub 2014 Jan 13.
4
A new human lung adenocarcinoma cell line harboring the EML4-ALK fusion gene.携带有 EML4-ALK 融合基因的新型人肺腺癌细胞系。
Jpn J Clin Oncol. 2014 Oct;44(10):963-8. doi: 10.1093/jjco/hyu110. Epub 2014 Aug 28.
5
Anaplastic lymphoma kinase rearrangement in lung cancer: its biological and clinical significance.肺癌中的间变性淋巴瘤激酶重排:其生物学及临床意义
Respir Investig. 2014 Nov;52(6):330-8. doi: 10.1016/j.resinv.2014.06.005. Epub 2014 Jul 30.
6
[Programs for continuing medical education: B session; 4. Progress in lung cancer therapy].[继续医学教育项目:B 环节;4. 肺癌治疗进展]
Nihon Naika Gakkai Zasshi. 2014 Mar 10;103(3):699-704. doi: 10.2169/naika.103.699.
7
[Lung adenocarcinoma with concomitant EGFR mutation and ALK rearrangement].伴有表皮生长因子受体(EGFR)突变和间变性淋巴瘤激酶(ALK)重排的肺腺癌
Rev Mal Respir. 2017 May;34(5):576-580. doi: 10.1016/j.rmr.2016.08.002. Epub 2016 Sep 17.
8
ALK fusion gene positive lung cancer and 3 cases treated with an inhibitor for ALK kinase activity.ALK 融合基因阳性肺癌及 3 例 ALK 激酶活性抑制剂治疗病例。
Lung Cancer. 2012 Jan;75(1):66-72. doi: 10.1016/j.lungcan.2011.05.027. Epub 2011 Jul 14.
9
ALK inhibitors in the treatment of advanced NSCLC.ALK 抑制剂治疗晚期 NSCLC。
Cancer Treat Rev. 2014 Mar;40(2):300-6. doi: 10.1016/j.ctrv.2013.07.002. Epub 2013 Aug 7.
10
Clinical outcomes in ALK-rearranged lung adenocarcinomas according to ALK fusion variants.根据ALK融合变体的间变性淋巴瘤激酶(ALK)重排肺腺癌的临床结局
J Transl Med. 2016 Oct 19;14(1):296. doi: 10.1186/s12967-016-1061-z.

引用本文的文献

1
Overall survival with crizotinib and next-generation ALK inhibitors in ALK-positive non-small-cell lung cancer (IFCT-1302 CLINALK): a French nationwide cohort retrospective study.克唑替尼与新一代ALK抑制剂用于ALK阳性非小细胞肺癌的总生存期(IFCT-1302 CLINALK):一项法国全国队列回顾性研究
Oncotarget. 2017 Mar 28;8(13):21903-21917. doi: 10.18632/oncotarget.15746.
2
Oncogenic kinase fusions: an evolving arena with innovative clinical opportunities.致癌激酶融合:一个充满创新临床机遇的不断发展的领域。
Oncotarget. 2016 May 3;7(18):25064-86. doi: 10.18632/oncotarget.7853.
3
ALK and crizotinib: after the honeymoon…what else? Resistance mechanisms and new therapies to overcome it.
ALK 和克唑替尼:蜜月之后……还有什么?耐药机制与克服耐药的新疗法。
Transl Lung Cancer Res. 2014 Aug;3(4):250-61. doi: 10.3978/j.issn.2218-6751.2014.03.01.
4
Tyrosine kinase inhibitors re-treatment beyond progression: choice and challenge.酪氨酸激酶抑制剂进展后再治疗:选择与挑战。
J Thorac Dis. 2014 Jun;6(6):595-7. doi: 10.3978/j.issn.2072-1439.2014.04.36.
5
Crizotinib: a review of its use in the treatment of anaplastic lymphoma kinase-positive, advanced non-small cell lung cancer.克唑替尼:治疗间变性淋巴瘤激酶阳性的晚期非小细胞肺癌的应用评价。
Drugs. 2013 Dec;73(18):2031-51. doi: 10.1007/s40265-013-0142-z.