Iga K, Kadowaki N, Hori K, Matumura T, Tamamura T, Tomonaga G, Gen H, Takahashi S
Department of Cardiology, Tenri Hospital.
Kokyu To Junkan. 1990 Jun;38(6):581-4.
We studied five cases of atrial fibrillation in the elderly showing severely decreased exercise tolerance (NYHA III) and normal left ventricular function. There were no signs of classical tricuspid regurgitation. Both atria were enlarged and right ventricular sizes were shown by two-dimensional echocardiography to have mildly increased. Two-dimensional Doppler echocardiography revealed severe tricupid regurgitation but at a maximal velocity less than 2.3 m/sec, meaning that right ventricular pressure was not increased. Cardiac output (CO) and circulating blood volume (BV), measured by radiocardiography showed CO to have decreased and BV to have increased. No intracardiac shunt was detected. The cause of this decreased exercise tolerance in the group in our study is thought to be right ventricular dysfunction due to the aging process. This also occurs in a similar pattern in younger people who have arrhythmogenic right ventricular dysplasia (ARVD), Uhl's disease, or right ventricular dilated cardiomyopathy.
我们研究了5例老年心房颤动患者,这些患者运动耐量严重下降(纽约心脏协会III级)且左心室功能正常。没有典型三尖瓣反流的迹象。两个心房均增大,二维超声心动图显示右心室大小轻度增加。二维多普勒超声心动图显示严重三尖瓣反流,但最大流速小于2.3米/秒,这意味着右心室压力未升高。通过放射性心动图测量的心输出量(CO)和循环血容量(BV)显示CO下降而BV增加。未检测到心内分流。我们研究组中运动耐量下降的原因被认为是由于衰老过程导致的右心室功能障碍。这在患有致心律失常性右心室发育不良(ARVD)、乌尔病或右心室扩张型心肌病的年轻人中也以类似模式发生。