Pigolkin Iu I, Dubrovina I A, Dubrovin I A, Khachaturian B S
Sud Med Ekspert. 2013 Mar-Apr;56(2):22-6.
The processes associated with liver disruption caused by a blunt abdominal injury have been investigated on a model of continuous media deformation. It was shown that local primary injuries to the liver result from stress-induced tissue stretching, compression, and shear in the longitudinal direction. They have a layered relief due to transverse fissures. The peripheral ruptures result from tissue extension under effect of overall liver deformation. They are not deep and have a uniform relief created by alternating low tubercles and superficial fissures oriented at the right angle to the surface. Antishock ruptures are the consequences of tissue extension or stretching/compression during local and overall deformation of the liver surface. They develop in the longitudinal direction, have a non-uniform relief created by alternating high prominences and numerous fissures differing in both the depth and the extension and oriented at the right angle to the liver surface. Central ruptures result from tissue stretching accompanying overall deformation of the organ. They are directed perpendicular to the direction of the surface force, have the longitudinal direction and non-uniform relief.
在连续介质变形模型上，对钝性腹部损伤所致肝脏破裂相关过程进行了研究。结果表明，肝脏局部原发性损伤是由应力诱导的组织纵向拉伸、压缩和剪切所致。由于横向裂隙，其具有分层的形态。周边破裂是肝脏整体变形作用下组织伸展的结果。它们不深，具有由交替出现的低结节和与表面成直角的浅表裂隙形成的均匀形态。抗休克破裂是肝脏表面局部和整体变形过程中组织伸展或拉伸/压缩的结果。它们沿纵向发展，具有由交替出现的高隆起和许多深度和伸展程度不同且与肝脏表面成直角的裂隙形成的不均匀形态。中央破裂是器官整体变形时组织拉伸的结果。它们垂直于表面力的方向，呈纵向且形态不均匀。
