Nakajima Y, Homma S, Musha T, Okamoto Y, Ackerman R H, Correia J A, Alpert N M
Department of Physiology, School of Medicine, Chiba University, Japan.
Neurosci Lett. 1990 Apr 20;112(1):59-64. doi: 10.1016/0304-3940(90)90322-z.
A patient with major neurological deficits 5 years after a left cerebral infarction underwent correlative EEG, MRI and PET studies of cerebral blood flow and oxygen metabolism. The EEG showed abnormal slow electroencephalographic activity in the frontopolar region. The intracranial location of the slow electrical activity was estimated, as an equivalent current dipole, by using a newly developed dipole tracing (DT) method. The DT analysis showed that the dipole equivalent of the slow wave is approximately located at the frontal part of the left cingulate gyrus, away from the margins of the infarction and enlarged left lateral ventricle demonstrated by MRI, and in a region with intact oxygen consumption rate. The genesis of the slow wave is discussed.
一名左侧脑梗死5年后出现严重神经功能缺损的患者接受了相关的脑电图（EEG）、磁共振成像（MRI）以及脑血流和氧代谢的正电子发射断层扫描（PET）研究。脑电图显示额极区存在异常的慢波脑电图活动。采用一种新开发的偶极子追踪（DT）方法，将慢电活动的颅内位置估计为等效电流偶极子。DT分析表明，慢波的等效偶极子大致位于左侧扣带回前部，远离MRI显示的梗死边缘和扩大的左侧脑室，且位于氧消耗率正常的区域。文中讨论了慢波的起源。
