Camac A, Greene P, Khandji A
Neurological Institute, Columbia Presbyterian Medical Center, New York, New York.
Mov Disord. 1990;5(3):235-8. doi: 10.1002/mds.870050309.
We describe a patient with the development of paroxysmal kinesigenic dystonic choreoathetosis (PKDC) after a thalamic infarct. PKDC consists of brief episodes of dystonia or choreoathetosis triggered by movement. PKDC improves with anticonvulsants, and in some cases, with L-Dopa or anticholinergics. We review PKDC, and relate its salient features to idiopathic and secondary torsion dystonia. We postulate a similar underlying pathophysiology.