Savel'ev S V, Besova N V
Ontogenez. 1990 May-Jun;21(3):298-303.
Portions of homologous neuroepithelium of various length were introduced into the neural tube cavity in Rana arvalis and Pleurodeles waltlii late neurula. The neural tube in R. arvalis was destroyed independently of the transplant length. In P. waltlii neural primordia were deformed if a large transplant prevented closing of the neural tube. Redundant increase in the host cells length (polarization) was observed during the small transplant-induced retardation of neurulation. Recipient cells elongated 1.5-2 fold as compared to the normal ones. Zones of contact polarization were detected. They arise in slowly relaxing layer under conditions during the transplant-induced retardation of neurulation. The labelled TRITC cells introduced into the neural tube cavity integrated into the wall of the neural tube and retarded polarization of its cells.
将不同长度的同源神经上皮部分植入湖蛙和肋突螈晚期神经胚的神经管腔内。湖蛙的神经管破坏与移植长度无关。在肋突螈中，如果大的移植块阻止神经管闭合，神经原基就会变形。在小移植块诱导神经胚形成延迟期间，观察到宿主细胞长度的冗余增加（极化）。与正常细胞相比，受体细胞伸长了1.5至2倍。检测到接触极化区。它们在移植诱导神经胚形成延迟期间的条件下，在缓慢松弛层中出现。引入神经管腔的标记TRITC细胞整合到神经管壁中，并延迟其细胞的极化。
