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心电图在识别心动过速性心肌病(TIC)病因方面的作用。

The ECG role in identifying the etiology of tachycardia-induced cardiomyopathy (TIC).

作者信息

Al Mehairi M, Al Ghamdi S A, Dagriri K, Al Fagih A

机构信息

Department of Adult Cardiology, Prince Sultan Cardiac Centre, Riyadh, Saudi Arabia.

出版信息

J Saudi Heart Assoc. 2012 Apr;24(2):133-6. doi: 10.1016/j.jsha.2011.10.008. Epub 2011 Dec 10.

DOI:10.1016/j.jsha.2011.10.008
PMID:23960682
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3727450/
Abstract

Tachycardia-induced cardiomyopathy (TIC) is a well recognized entity of heart failure (HF) and various mechanisms due to tachyarrhythmias have been postulated to be responsible for impaired cardiac contractility. Previously reported cases showed reversibility of such disorders whenever stable cardiac rhythm is maintained adequately and we report on a 16-year-old boy who has been diagnosed to have TIC, which was misinterpreted initially as sinus tachycardia secondary to dilated cardiomyopathy and heart failure. A complete recovery of his left ventricular function was achieved by radiofrequency catheter ablation and highlights the importance of a 12-lead electrocardiogram (ECG) assessment in such patients.

摘要

心动过速性心肌病(TIC)是一种已被充分认识的心力衰竭(HF)类型,据推测,各种由快速性心律失常引起的机制与心脏收缩功能受损有关。先前报道的病例显示,只要能充分维持稳定的心律,此类疾病具有可逆性。我们报告了一名16岁男孩,他被诊断患有TIC,最初被误诊为继发于扩张型心肌病和心力衰竭的窦性心动过速。通过射频导管消融,他的左心室功能完全恢复,这突出了对此类患者进行12导联心电图(ECG)评估的重要性。

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本文引用的文献

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Tachycardia-induced cardiomyopathy: mechanisms of heart failure and clinical implications.心动过速性心肌病:心力衰竭的机制及临床意义
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Imbalance between xanthine oxidase and nitric oxide synthase signaling pathways underlies mechanoenergetic uncoupling in the failing heart.黄嘌呤氧化酶与一氧化氮合酶信号通路之间的失衡是衰竭心脏机械能量解偶联的基础。
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Reversal of tachycardia induced cardiomyopathy following ablation of repetitive monomorphic right ventricular outflow tract tachycardia.重复性单形性右心室流出道心动过速消融术后心动过速性心肌病的逆转
Pacing Clin Electrophysiol. 2001 Feb;24(2):166-71. doi: 10.1046/j.1460-9592.2001.00166.x.
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Reduction in density of transverse tubules and L-type Ca(2+) channels in canine tachycardia-induced heart failure.犬心动过速诱导性心力衰竭时横小管和L型钙通道密度降低。
Cardiovasc Res. 2001 Feb 1;49(2):298-307. doi: 10.1016/s0008-6363(00)00256-x.
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Tachycardia-related cardiomyopathy: a common cause of ventricular dysfunction in patients with atrial fibrillation referred for atrioventricular ablation.
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Rapid electrical stimulation of contraction reduces the density of beta-adrenergic receptors and responsiveness of cultured neonatal rat cardiomyocytes. Possible involvement of microtubule disassembly secondary to mechanical stress.快速电刺激收缩可降低培养的新生大鼠心肌细胞β-肾上腺素能受体的密度和反应性。可能涉及机械应激继发的微管解聚。
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First evidence of premature ventricular complex-induced cardiomyopathy: a potentially reversible cause of heart failure.室性早搏诱发心肌病的首个证据:心力衰竭的一个潜在可逆病因。
J Cardiovasc Electrophysiol. 2000 Mar;11(3):328-9. doi: 10.1111/j.1540-8167.2000.tb01802.x.
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Left ventricular myocardial blood flow, metabolism, and effects of treatment with enalapril: further insights into the mechanisms of canine experimental pacing-induced heart failure.左心室心肌血流、代谢及依那普利治疗的效果:对犬实验性起搏诱导心力衰竭机制的进一步认识
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