Chang Tzu-Pu, Wu Yi-Chang, Hsu Yung-Chu
Department of Neurology, Neuro-Medical Scientific Center, Buddhist Tzu Chi General Hospital, Taichung Branch, Taiwan.
Acta Neurol Taiwan. 2013 Jun;22(2):72-5.
Vestibular paroxysmia is defined as paroxysmal, brief, and carbamazepine-responsive vertigo. Although neurovascular cross-compression (NVCC) of the vestibulocochlear nerve is believed to be the cause of vestibular paroxysmia, the mechanism remains controversial. Herein, we describe the case of a man with NVCC who presented with paroxysmal vertigo associated with paroxysmal pulsatile tinnitus.
A 68-year-old man presented with paroxysmal vertigo for one month. Paroxysmal pulsatile tinnitus in the right ear occurred simultaneously with the vertigo. Magnetic resonance imaging demonstrated that the right anterior inferior cerebellar artery was compressing the right vestibulocochlear nerve. The vertigo and tinnitus completely disappeared within one week after treatment with carbamazepine.
The pulsatile nature of the patient's tinnitus implied that the auditory nerve was being compressed by a pulsating artery and was found to consolidate the causal relationship between NVCC and vestibular paroxysmia.
前庭阵发性眩晕被定义为阵发性、短暂性且对卡马西平有反应的眩晕。尽管听神经的神经血管交叉压迫(NVCC)被认为是前庭阵发性眩晕的病因,但其机制仍存在争议。在此,我们描述了一例患有NVCC且伴有阵发性眩晕和阵发性搏动性耳鸣的男性病例。
一名68岁男性出现阵发性眩晕1个月。眩晕发作时,右耳同时出现阵发性搏动性耳鸣。磁共振成像显示,右小脑前下动脉压迫右听神经。使用卡马西平治疗1周内,眩晕和耳鸣完全消失。
患者耳鸣的搏动性质表明听觉神经受到搏动性动脉的压迫,这证实了NVCC与前庭阵发性眩晕之间的因果关系。