• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Immunopathologic events at the endplate in myasthenia gravis.

作者信息

Ashizawa T, Appel S H

出版信息

Springer Semin Immunopathol. 1985;8(3):177-96. doi: 10.1007/BF00197295.

DOI:10.1007/BF00197295
PMID:2413561
Abstract
摘要

相似文献

1
Immunopathologic events at the endplate in myasthenia gravis.重症肌无力终板处的免疫病理事件。
Springer Semin Immunopathol. 1985;8(3):177-96. doi: 10.1007/BF00197295.
2
Passive transfer of myasthenia gravis by immunoglobulins: lack of correlation between AChR with antibody bound, acetylcholine receptor loss and transmission defect.重症肌无力通过免疫球蛋白的被动转移：与抗体结合的乙酰胆碱受体、乙酰胆碱受体丧失和传递缺陷之间缺乏相关性。
J Neurol Sci. 1988 Mar;84(1):15-28. doi: 10.1016/0022-510x(88)90170-0.
3
Experimental myasthenia: lack of correlation between the autoantibody titer and the reduction of acetylcholine-controlled ionic channels measured at functioning endplates.
Muscle Nerve. 1983 Feb;6(2):160-3. doi: 10.1002/mus.880060213.
4
The membrane attack complex of complement at the endplate in myasthenia gravis.重症肌无力终板处补体的膜攻击复合物
Ann N Y Acad Sci. 1987;505:326-32. doi: 10.1111/j.1749-6632.1987.tb51301.x.
5
[Clinical significance of immune complexes at the motor endplate in myasthenia gravis].[重症肌无力运动终板处免疫复合物的临床意义]
Rinsho Shinkeigaku. 1983 Sep;23(9):735-43.
6
Increased uptake of acetylcholine-receptor antibody at motor endplate in myasthenic exacerbation.重症肌无力加重期运动终板处乙酰胆碱受体抗体摄取增加。
Lancet. 1978 Jan 7;1(8054):47. doi: 10.1016/s0140-6736(78)90396-3.
7
The immunopathological basis of acetylcholine receptor deficiency in myasthenia gravis.重症肌无力中乙酰胆碱受体缺乏的免疫病理学基础。
Prog Brain Res. 1979;49:423-34. doi: 10.1016/S0079-6123(08)64654-3.
8
Pre- and postsynaptic neuromuscular junction abnormalities in musk myasthenia.先天性肌无力症的突触前和突触后神经肌肉接头异常。
Muscle Nerve. 2010 Aug;42(2):283-8. doi: 10.1002/mus.21642.
9
Myasthenia Gravis.重症肌无力
Mayo Clin Proc. 1977 May;52(5):334-5.
10
Structure-function correlations in myasthenia gravis and a new myasthenic syndrome.重症肌无力及一种新型肌无力综合征的结构-功能相关性
Electroencephalogr Clin Neurophysiol Suppl. 1978(34):469-77.

引用本文的文献

1
Emerging role of complement system in the induction of neuroinflammation in adenylosuccinate lyase deficiency disorder.腺苷酸琥珀酸裂解酶缺乏症中补体系统在诱导神经炎症方面的新作用。
Brain Behav Immun Health. 2025 Aug 19;48:101091. doi: 10.1016/j.bbih.2025.101091. eCollection 2025 Oct.
2
Hypocomplementemia as a Risk Factor for Organ Damage Accrual in Patients with Systemic Lupus Erythematosus.低补体血症作为系统性红斑狼疮患者器官损害累积的风险因素。
J Immunol Res. 2018 Dec 30;2018:8051972. doi: 10.1155/2018/8051972. eCollection 2018.
3
The role of complement in experimental autoimmune myasthenia gravis.

本文引用的文献

1
AN ELECTROPHYSIOLOGICAL INVESTIGATION OF NEUROMUSCULAR TRANSMISSION IN MYASTHENIA GRAVIS.重症肌无力神经肌肉传递的电生理研究
J Physiol. 1964 Nov;174(3):417-34. doi: 10.1113/jphysiol.1964.sp007495.
2
Changes in serum complement activity in patients with myasthenia gravis.重症肌无力患者血清补体活性的变化
Proc Soc Exp Biol Med. 1960 Oct;105:177-84. doi: 10.3181/00379727-105-26050.
3
Histological changes in the striped muscles in myasthenia gravis.重症肌无力患者横纹肌的组织学变化。
补体在实验性自身免疫性重症肌无力中的作用。
Ann N Y Acad Sci. 2012 Dec;1274(1):127-32. doi: 10.1111/j.1749-6632.2012.06783.x.
4
Complement and systemic lupus erythematosus.补体与系统性红斑狼疮
Arthritis Res. 2002;4 Suppl 3(Suppl 3):S279-93. doi: 10.1186/ar586. Epub 2002 May 9.
5
Immunopathogenesis and treatment of myasthenia gravis.重症肌无力的免疫发病机制与治疗
J Clin Immunol. 1987 May;7(3):187-97. doi: 10.1007/BF00915723.
6
Phenotypic characteristics of thymic B lymphocytes in myasthenia gravis.
J Clin Immunol. 1989 May;9(3):242-7. doi: 10.1007/BF00916820.
7
Epitope-specific suppression of antibody response in experimental autoimmune myasthenia gravis by a monomethoxypolyethylene glycol conjugate of a myasthenogenic synthetic peptide.通过致重症肌无力合成肽的单甲氧基聚乙二醇缀合物对实验性自身免疫性重症肌无力抗体反应进行表位特异性抑制。
Proc Natl Acad Sci U S A. 1992 Jul 1;89(13):5852-6. doi: 10.1073/pnas.89.13.5852.
J Pathol Bacteriol. 1953 Apr;65(2):279-89. doi: 10.1002/path.1700650202.
4
Monoclonal antibodies directed against the neurotransmitter binding site of nicotinic acetylcholine receptor.针对烟碱型乙酰胆碱受体神经递质结合位点的单克隆抗体。
FEBS Lett. 1980 Oct 20;120(1):145-8. doi: 10.1016/0014-5793(80)81066-0.
5
Monoclonal anti-acetylcholine receptor antibodies can cause experimental myasthenia.
Nature. 1980 Aug 14;286(5774):738-9. doi: 10.1038/286738a0.
6
Diffusion of acetylcholine in the synaptic cleft of normal and myasthenia gravis human endplates.乙酰胆碱在正常人和重症肌无力患者终板突触间隙中的扩散。
Nature. 1980 Jul 31;286(5772):500-2. doi: 10.1038/286500a0.
7
Anti-acetylcholine receptor antibodies.抗乙酰胆碱受体抗体
J Neurol Neurosurg Psychiatry. 1980 Jul;43(7):590-600. doi: 10.1136/jnnp.43.7.590.
8
Ultrastructural localization of the terminal and lytic ninth complement component (C9) at the motor end-plate in myasthenia gravis.重症肌无力运动终板处终末溶解型第九补体成分（C9）的超微结构定位
J Neuropathol Exp Neurol. 1980 Mar;39(2):160-72. doi: 10.1097/00005072-198003000-00005.
9
Degradation of the acetylcholine receptor in cultured muscle cells: selective inhibitors and the fate of undegraded receptors.培养的肌细胞中乙酰胆碱受体的降解：选择性抑制剂及未降解受体的命运
Cell. 1980 Feb;19(2):481-91. doi: 10.1016/0092-8674(80)90523-1.
10
Experimental myasthenia gravis. A murine system.实验性重症肌无力。一种小鼠模型系统。
J Exp Med. 1980 Jan 1;151(1):204-23. doi: 10.1084/jem.151.1.204.