相似文献
1
No antigen-presentation defect in Unc93b1(3d/3d) (3d) mice.Unc93b1(3d/3d)（3d）小鼠不存在抗原呈递缺陷。
Nat Immunol. 2013 Nov;14(11):1101-2. doi: 10.1038/ni.2733.
2
Respond to "No antigen-presentation defect in Unc93b1(3d/3d) (3d) mice".回应“Unc93b1(3d/3d) (3d)小鼠无抗原呈递缺陷”。
Nat Immunol. 2013 Nov;14(11):1102-3. doi: 10.1038/ni.2734.
3
The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9.Unc93b1突变3d通过Toll样受体3、7和9破坏外源性抗原呈递和信号传导。
Nat Immunol. 2006 Feb;7(2):156-64. doi: 10.1038/ni1297. Epub 2006 Jan 15.
4
UNC93B1 and nucleic acid-sensing Toll-like receptors mediate host resistance to infection with Leishmania major.UNC93B1 和核酸感应 Toll 样受体介导宿主对感染利什曼原虫的抗性。
J Biol Chem. 2013 Mar 8;288(10):7127-36. doi: 10.1074/jbc.M112.407684. Epub 2013 Jan 16.
5
Unc93B1 restricts systemic lethal inflammation by orchestrating Toll-like receptor 7 and 9 trafficking.UNC93B1 通过协调 Toll 样受体 7 和 9 的运输来限制全身性致死性炎症。
Immunity. 2011 Jul 22;35(1):69-81. doi: 10.1016/j.immuni.2011.05.010. Epub 2011 Jun 16.
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UNC93B1 delivers nucleotide-sensing toll-like receptors to endolysosomes.UNC93B1将核苷酸感应型 toll 样受体递送至内溶酶体。
Nature. 2008 Mar 13;452(7184):234-8. doi: 10.1038/nature06726. Epub 2008 Feb 27.
7
Release from UNC93B1 reinforces the compartmentalized activation of select TLRs.UNC93B1 的释放增强了特定 TLR 的区室化激活。
Nature. 2019 Nov;575(7782):371-374. doi: 10.1038/s41586-019-1611-7. Epub 2019 Sep 23.
8
Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to infection.巨噬细胞内体Toll样受体信号下游的自噬是抵抗感染的关键机制。
J Biol Chem. 2017 Aug 11;292(32):13087-13096. doi: 10.1074/jbc.M117.780981. Epub 2017 Jun 12.
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The interaction between the ER membrane protein UNC93B and TLR3, 7, and 9 is crucial for TLR signaling.内质网（ER）膜蛋白UNC93B与Toll样受体（TLR）3、7和9之间的相互作用对于TLR信号传导至关重要。
J Cell Biol. 2007 Apr 23;177(2):265-75. doi: 10.1083/jcb.200612056.
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Unc93B1 biases Toll-like receptor responses to nucleic acid in dendritic cells toward DNA- but against RNA-sensing.Unc93B1使树突状细胞中Toll样受体对核酸的反应偏向于识别DNA而非RNA。
J Exp Med. 2009 Jun 8;206(6):1339-50. doi: 10.1084/jem.20082316. Epub 2009 May 18.
引用本文的文献
1
Current Concepts of Antigen Cross-Presentation.抗原交叉呈递的当前概念
Front Immunol. 2018 Jul 16;9:1643. doi: 10.3389/fimmu.2018.01643. eCollection 2018.
2
Cross-Presentation of Cell-Associated Antigens by MHC Class I in Dendritic Cell Subsets.树突状细胞亚群中MHC I类分子对细胞相关抗原的交叉呈递
Front Immunol. 2015 Jul 17;6:363. doi: 10.3389/fimmu.2015.00363. eCollection 2015.
3
Unc93b1 -Dependent Endosomal Toll-Like Receptor Signaling Regulates Inflammation and Mortality during Coxsackievirus B3 Infection.Unc93b1依赖性内体 Toll 样受体信号传导调节柯萨奇病毒B3感染期间的炎症和死亡率。
J Innate Immun. 2015;7(3):315-30. doi: 10.1159/000369342. Epub 2015 Feb 11.
4
Toll-like receptor-deficient mice reveal how innate immune signaling influences Salmonella virulence strategies.Toll 样受体缺陷小鼠揭示了固有免疫信号如何影响沙门氏菌毒力策略。
Cell Host Microbe. 2014 Feb 12;15(2):203-13. doi: 10.1016/j.chom.2014.01.013.
本文引用的文献
1
Acidic amino acid residues in the juxtamembrane region of the nucleotide-sensing TLRs are important for UNC93B1 binding and signaling.核苷酸感应 TLR 近膜区的酸性氨基酸残基对于 UNC93B1 的结合和信号转导很重要。
J Immunol. 2013 May 15;190(10):5287-95. doi: 10.4049/jimmunol.1202767. Epub 2013 Apr 12.
2
UNC93B1 mediates differential trafficking of endosomal TLRs.UNC93B1介导内体Toll样受体的差异运输。
Elife. 2013 Feb 19;2:e00291. doi: 10.7554/eLife.00291.
3
TLR13 recognizes bacterial 23S rRNA devoid of erythromycin resistance-forming modification.TLR13 识别缺乏红霉素耐药形成修饰的细菌 23S rRNA。
Science. 2012 Aug 31;337(6098):1111-5. doi: 10.1126/science.1220363. Epub 2012 Jul 19.
4
UNC93B1 delivers nucleotide-sensing toll-like receptors to endolysosomes.UNC93B1将核苷酸感应型 toll 样受体递送至内溶酶体。
Nature. 2008 Mar 13;452(7184):234-8. doi: 10.1038/nature06726. Epub 2008 Feb 27.
5
Assessing the function of human UNC-93B in Toll-like receptor signaling and major histocompatibility complex II response.评估人类UNC-93B在Toll样受体信号传导和主要组织相容性复合体II反应中的功能。
Hum Immunol. 2007 Nov;68(11):871-8. doi: 10.1016/j.humimm.2007.07.007. Epub 2007 Sep 24.
6
Herpes simplex virus encephalitis in human UNC-93B deficiency.人类UNC-93B缺陷型单纯疱疹病毒性脑炎。
Science. 2006 Oct 13;314(5797):308-12. doi: 10.1126/science.1128346. Epub 2006 Sep 14.
7
The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9.Unc93b1突变3d通过Toll样受体3、7和9破坏外源性抗原呈递和信号传导。
Nat Immunol. 2006 Feb;7(2):156-64. doi: 10.1038/ni1297. Epub 2006 Jan 15.
8
Toll-like receptor control of the adaptive immune responses.Toll样受体对适应性免疫反应的调控。
Nat Immunol. 2004 Oct;5(10):987-95. doi: 10.1038/ni1112.
9
The effect of mutations in the MHC class I peptide binding groove on the cytotoxic T lymphocyte recognition of the Kb-restricted ovalbumin determinant.MHC I类肽结合槽中的突变对细胞毒性T淋巴细胞对Kb限制性卵清蛋白决定簇识别的影响。
Eur J Immunol. 1990 Nov;20(11):2431-7. doi: 10.1002/eji.1830201111.
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