Lv Xianli, Li Youxiang, Jiang Chuhan, Zhang Jingbo, Wu Zhongxue
Beijing Neurosurgical Institute and Beijing Tiantan Hospital, Capital Medical University; Beijing, China -
Neuroradiol J. 2008 Oct 1;21(4):579-83. doi: 10.1177/197140090802100418.
Dural arteriovenous fistulas with restricted antegrade venous flow and reflux into a dural sinus or cortical veins lead to intracranial venous hypertension. This type of venous hypertension in return may cause parenchymal damage by mechanisms similar to those proposed for venous infarction. A 42-year-old man presented with homonymous hemianopsia of the right upper visual field. Computed tomography demonstrated hypointensity in the left occipital lobe. Angiography confirmed dural arteriovenous fistula of the left sigmoid sinus, severe stenosis and occlusion of the left sigmoid sinus and reversal of the flow in the cortical veins. This fistula was treated with Onyx-18. This report emphasizes an unusual coexistence of venous infarction with sigmoid sinus dural arteriovenous fistula causing neurological symptoms.
伴有顺行性静脉血流受限及血液反流至硬脑膜窦或皮质静脉的硬脑膜动静脉瘘会导致颅内静脉高压。这种类型的静脉高压反过来可能通过与静脉梗死所提出的机制相似的机制导致实质损伤。一名42岁男性出现右上视野同向性偏盲。计算机断层扫描显示左侧枕叶呈低密度。血管造影证实左侧乙状窦硬脑膜动静脉瘘、左侧乙状窦严重狭窄和闭塞以及皮质静脉血流逆转。该瘘用Onyx - 18进行了治疗。本报告强调了静脉梗死与导致神经症状的乙状窦硬脑膜动静脉瘘异常并存的情况。