Station Biologique de Beau-Site, University of Poitiers, 25 Faubourg Saint Cyprien, F-86000, Poitiers, France.
Planta. 1984 Feb;160(2):109-12. doi: 10.1007/BF00392858.
Prostaglandin E1 and prostaglandin E2 speed up the dark-induced (scotonastic) and light-induced (photonastic) leaflet movements of Cassia fasciculata. The precursors of prostaglandin biosynthesis, homo γ-linolenic and arachidonic acids, and an intermediary product, prostaglandin-interm-5, act in the same manner on these movements. Inhibitors of prostaglandin biosynthesis, indomethacin and phenylbutazone, inhibited the scotonastic but promoted the photonastic movements in an unexpected way. Since the pulvinar movements are mediated by water and ion migrations, the observed modifications of these movements indicate that prostaglandins and their precursors may affect, as in animal cells, processes linked to a variation of membrane permeability.
前列腺素 E1 和前列腺素 E2 加速了决明子的暗诱导（暗向性）和光诱导（光向性）叶片运动。前列腺素生物合成的前体，同型 γ-亚麻酸和花生四烯酸，以及中间产物，前列腺素-interm-5，以相同的方式作用于这些运动。前列腺素生物合成的抑制剂，吲哚美辛和保泰松，以出人意料的方式抑制了暗向性运动，却促进了光向性运动。由于叶枕运动是由水和离子迁移介导的，因此这些运动的观察到的变化表明，前列腺素及其前体可能会影响，就像在动物细胞中一样，与膜通透性变化相关的过程。
