Zimmern S H, Clark M F, Austin W K, Fedor J M, Gallagher J J, Svenson R H, Duncan J L
Pacing Clin Electrophysiol. 1986 Nov;9(6):1019-25. doi: 10.1111/j.1540-8159.1986.tb06664.x.
Myopotential signals were recorded from atrial and ventricular leads during isometric exercise in 25 patients who had chronically implanted dual chamber pacemakers using the electrogram telemetry capability of the pacemakers. Average electrogram amplitude on the atrial channel was 0.92 mV (range 0.3 to 1.9) and on the ventricular channel was 0.98 mV (range 0.3 to 2.2); the difference was not significant. There was a strong correlation (R = 0.82) between the amplitude of myopotentials on the atrial and ventricular leads for individual patients. Myopotential sensing caused ventricular output inhibition in two patients (8%) and ventricular tracking in sixteen patients (64%). Pacemaker reprogramming abolished ventricular myopotential inhibition in all patients and stopped ventricular myopotential tracking in seven patients. We conclude that myopotentials can be analyzed and their effects ameliorated by a multiprogrammable pacemaker with electrogram telemetry capability.
在25例长期植入双腔起搏器的患者进行等长运动期间，利用起搏器的心电图遥测功能，从心房和心室导联记录肌电位信号。心房通道的平均心电图振幅为0.92mV（范围0.3至1.9），心室通道为0.98mV（范围0.3至2.2）；差异不显著。个体患者心房和心室导联上肌电位振幅之间存在强相关性（R = 0.82）。肌电位感知导致2例患者（8%）出现心室输出抑制，16例患者（64%）出现心室跟踪。起搏器重新编程消除了所有患者的心室肌电位抑制，并使7例患者的心室肌电位跟踪停止。我们得出结论，具有心电图遥测功能的多程控起搏器可以分析肌电位并改善其影响。
