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Semaphorins as Potential Immune Therapeutic Targets for Cancer.
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Tumor hypoxia does not drive differentiation of tumor-associated macrophages but rather fine-tunes the M2-like macrophage population.
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Low-dose irradiation programs macrophage differentiation to an iNOS⁺/M1 phenotype that orchestrates effective T cell immunotherapy.
Cancer Cell. 2013 Nov 11;24(5):589-602. doi: 10.1016/j.ccr.2013.09.014. Epub 2013 Oct 24.
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Macrophage regulation of tumor responses to anticancer therapies.
Cancer Cell. 2013 Mar 18;23(3):277-86. doi: 10.1016/j.ccr.2013.02.013.
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Neutralizing tumor-promoting chronic inflammation: a magic bullet?
Science. 2013 Jan 18;339(6117):286-91. doi: 10.1126/science.1232227.
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The good and bad of microglia/macrophages: new hope in stroke therapeutics.
Acta Pharmacol Sin. 2013 Jan;34(1):6-7. doi: 10.1038/aps.2012.178. Epub 2012 Dec 24.
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Macrophage diversity enhances tumor progression and metastasis.
Cell. 2010 Apr 2;141(1):39-51. doi: 10.1016/j.cell.2010.03.014.
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The Yin-Yang of tumor-associated macrophages in neoplastic progression and immune surveillance.
Immunol Rev. 2008 Apr;222:155-61. doi: 10.1111/j.1600-065X.2008.00607.x.
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Blocking neuropilin-1 function has an additive effect with anti-VEGF to inhibit tumor growth.
Cancer Cell. 2007 Jan;11(1):53-67. doi: 10.1016/j.ccr.2006.10.018.

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