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Trafficking regulation of proteins in Alzheimer's disease.
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Regulation of Synaptic Amyloid-β Generation through BACE1 Retrograde Transport in a Mouse Model of Alzheimer's Disease.
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Molecular and cellular mechanisms for Alzheimer's disease: understanding APP metabolism.
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Cellular Trafficking of Amyloid Precursor Protein in Amyloidogenesis Physiological and Pathological Significance.
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The role of TMP21 in trafficking and amyloid-β precursor protein (APP) processing in Alzheimer's disease.
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Intracellular trafficking of the β-secretase and processing of amyloid precursor protein.
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2
Effects and mechanisms of APP and its cleavage product Aβ in the comorbidity of sarcopenia and Alzheimer's disease.
Front Aging Neurosci. 2024 Nov 25;16:1482947. doi: 10.3389/fnagi.2024.1482947. eCollection 2024.
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Endolysosomal trafficking controls yolk granule biogenesis in vitellogenic Drosophila oocytes.
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Amyloid Precursor Protein and Alzheimer's Disease.
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Polyphenols, Autophagy and Neurodegenerative Diseases: A Review.
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Ginsenoside ompound K reduces neuronal damage and improves neuronal synaptic dysfunction by targeting Aβ.
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Prenylomic Profiling in the Brain of a Transgenic Mouse Model of Alzheimer's Disease Reveals Increased Prenylation of a Key Set of Proteins.
ACS Chem Biol. 2022 Oct 21;17(10):2863-2876. doi: 10.1021/acschembio.2c00486. Epub 2022 Sep 15.
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Endosomal structure and APP biology are not altered in a preclinical mouse cellular model of Down syndrome.
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Aβ Assemblies Promote Amyloidogenic Processing of APP and Intracellular Accumulation of Aβ42 Through Go/Gβγ Signaling.
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Secretases Related to Amyloid Precursor Protein Processing.
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The role of reticulons in neurodegenerative diseases.
Neuromolecular Med. 2014 Mar;16(1):3-15. doi: 10.1007/s12017-013-8271-9. Epub 2013 Nov 12.
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Gpr3 stimulates Aβ production via interactions with APP and β-arrestin2.
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Elucidation of the BACE1 regulating factor GGA3 in Alzheimer's disease.
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Endocytosis of synaptic ADAM10 in neuronal plasticity and Alzheimer's disease.
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Apolipoprotein E and Alzheimer disease: risk, mechanisms and therapy.
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β-arrestin1 regulates γ-secretase complex assembly and modulates amyloid-β pathology.
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β-arrestin 2 regulates Aβ generation and γ-secretase activity in Alzheimer's disease.
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Ubiquilin-1 regulates amyloid precursor protein maturation and degradation by stimulating K63-linked polyubiquitination of lysine 688.
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