Hartshorn K L, Tauber A I
Boston University School of Medicine, Massachusetts.
Hematol Oncol Clin North Am. 1988 Jun;2(2):301-15.
Influenza A virus has the ability to activate human neutrophils and in turn reduce its subsequent oxidative, chemotactic, and degranulation responses to other stimuli, an effect that may lead to adverse clinical sequelae. Current literature on the mechanism of this effect is reviewed, and preliminary data regarding its biochemical basis presented.
甲型流感病毒能够激活人类中性粒细胞，进而降低其随后对其他刺激的氧化、趋化和脱颗粒反应，这种效应可能导致不良临床后果。本文综述了关于这种效应机制的现有文献，并给出了关于其生化基础的初步数据。
