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The role of PAX5 and C/EBP α/β in atypical non-Langerhans cell histiocytic tumor post acute lymphoblastic leukemia.

作者信息

Pagni F, Fazio G, Zannella S, Spinelli M, De Angelis C, Cusi C, Crosti F, Corral L, Bugarin C, Biondi A, Cazzaniga G, Isimbaldi G, Cattoretti G

机构信息

Department of Surgery and Interdisciplinary Medicine, Pathology Section, University Milano-Bicocca, Milan, Italy.

1] Centro Ricerca Tettamanti, Pediatric Clinic, University of Milan Bicocca, San Gerardo Hospital/Fondazione MBBM, Monza, Italy [2] Department of Health Sciences, University of Milan Bicocca, Monza, Italy.

出版信息

Leukemia. 2014 Jun;28(6):1377-9. doi: 10.1038/leu.2014.87. Epub 2014 Feb 25.

DOI:10.1038/leu.2014.87
PMID:24569775
Abstract
摘要

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The role of PAX5 and C/EBP α/β in atypical non-Langerhans cell histiocytic tumor post acute lymphoblastic leukemia.PAX5和C/EBPα/β在急性淋巴细胞白血病后非典型非朗格汉斯细胞组织细胞性肿瘤中的作用
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Altered mRNA expression of PAX5 is a common event in acute lymphoblastic leukaemia.PAX5的mRNA表达改变在急性淋巴细胞白血病中是常见现象。
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Mismatch repair and the downstream target genes, PAX5 and Ikaros, in childhood acute lymphoblastic leukemia.错配修复与下游靶基因 PAX5 和 Ikaros 在儿童急性淋巴细胞白血病中的作用。
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PAX5 P80R-mutated B-cell acute lymphoblastic leukemia with transformation to histiocytic sarcoma: clonal evolution assessment using NGS-based immunoglobulin clonality and mutation analysis.伴有 PAX5 P80R 突变的 B 细胞急性淋巴细胞白血病向组织细胞肉瘤转化：基于 NGS 的免疫球蛋白克隆性和突变分析的克隆进化评估。
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本文引用的文献

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CD2-positive B-cell precursor acute lymphoblastic leukemia with an early switch to the monocytic lineage.CD2 阳性 B 细胞前体急性淋巴细胞白血病向单核细胞系早期分化。
Leukemia. 2014 Mar;28(3):609-20. doi: 10.1038/leu.2013.354. Epub 2013 Nov 25.
2
Lymphoid to myeloid cell trans-differentiation is determined by C/EBPβ structure and post-translational modifications.淋巴样细胞向髓样细胞的转分化由 C/EBPβ 的结构和翻译后修饰决定。
PLoS One. 2013 Jun 5;8(6):e65169. doi: 10.1371/journal.pone.0065169. Print 2013.
3
Diverse and heritable lineage imprinting of early haematopoietic progenitors.
Independent development of lymphoid and histiocytic malignancies from a shared early precursor.
源自共同早期前体的淋巴样和组织细胞性恶性肿瘤的独立发生
Leukemia. 2016 Apr;30(4):955-8. doi: 10.1038/leu.2015.193. Epub 2015 Jul 23.
早期造血祖细胞的多样化和可遗传的谱系印记。
Nature. 2013 Apr 11;496(7444):229-32. doi: 10.1038/nature12013. Epub 2013 Apr 3.
4
C/EBPα induces highly efficient macrophage transdifferentiation of B lymphoma and leukemia cell lines and impairs their tumorigenicity.C/EBPα 诱导 B 淋巴瘤和白血病细胞系中高效的巨噬细胞转分化，并损害其致瘤性。
Cell Rep. 2013 Apr 25;3(4):1153-63. doi: 10.1016/j.celrep.2013.03.003. Epub 2013 Mar 28.
5
Dramatic efficacy of vemurafenib in both multisystemic and refractory Erdheim-Chester disease and Langerhans cell histiocytosis harboring the BRAF V600E mutation.携带 BRAF V600E 突变的多系统和难治性 Erdheim-Chester 病和朗格汉斯细胞组织细胞增生症中维莫非尼的显著疗效。
Blood. 2013 Feb 28;121(9):1495-500. doi: 10.1182/blood-2012-07-446286. Epub 2012 Dec 20.
6
B-lineage transcription factors and cooperating gene lesions required for leukemia development.B 细胞系转录因子和协同基因病变在白血病发生中的作用。
Leukemia. 2013 Mar;27(3):541-52. doi: 10.1038/leu.2012.293. Epub 2012 Oct 10.
7
Biphenotypic B-lymphoid/myeloid cells expressing low levels of Pax5: potential targets of BAL development.表达低水平 Pax5 的双表型 B 淋巴细胞/髓系细胞：BAL 发育的潜在靶点。
Blood. 2012 Nov 1;120(18):3688-98. doi: 10.1182/blood-2012-03-414821. Epub 2012 Aug 27.
8
Molecular response to treatment redefines all prognostic factors in children and adolescents with B-cell precursor acute lymphoblastic leukemia: results in 3184 patients of the AIEOP-BFM ALL 2000 study.治疗后的分子反应重新定义了儿童和青少年 B 细胞前体急性淋巴细胞白血病的所有预后因素：AIEOP-BFM ALL 2000 研究 3184 例患者的结果。
Blood. 2010 Apr 22;115(16):3206-14. doi: 10.1182/blood-2009-10-248146. Epub 2010 Feb 12.
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Clinicopathologic features of histiocytic lesions following ALL, with a review of the literature.急性淋巴细胞白血病后组织细胞病变的临床病理特征，并附文献综述。
Pediatr Dev Pathol. 2010 May-Jun;13(3):225-37. doi: 10.2350/09-03-0622-OA.1.
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Genomic analysis of the clonal origins of relapsed acute lymphoblastic leukemia.复发性急性淋巴细胞白血病克隆起源的基因组分析
Science. 2008 Nov 28;322(5906):1377-80. doi: 10.1126/science.1164266.