Sato Junko, Ishii Yoshinori, Noguchi Hideo, Takeda Mitsuhiro
Ishii Orthopaedic & Rehabilitation Clinic, Gyoda, Saitama, Japan.
Ishii Orthopaedic & Rehabilitation Clinic, Gyoda, Saitama, Japan.
Ultrasound Med Biol. 2014 Jun;40(6):1146-53. doi: 10.1016/j.ultrasmedbio.2014.01.009. Epub 2014 Mar 6.
This study investigated the sonographic appearance of the pulley and flexor tendon in idiopathic trigger finger in correlation with the contracture of the interphalangeal (IP) joint in the thumb or proximal IP (PIP) joint in the other digits. Sonographic measurements using axial images were performed in 177 affected digits including 17 thumbs and 34 other digits judged to have IP or PIP joint contracture and 77 contralateral control digits. The A1 pulley of the contracture group was significantly thicker than that of the non-contracture group in all digits, whereas the flexor tendon was thicker only in digits other than the thumb. In the analysis using calculated cut-off values, A1 pulley thickening in the thumb and A1 pulley and flexor tendon thickening in the other digits showed statistically significant correlations with IP or PIP joint contracture. This study sonographically confirmed previous reports showing that enlargement of the flexor tendons contribute to the pathogenesis of PIP joint contracture.
本研究调查了特发性扳机指中滑车和屈肌腱的超声表现，并将其与拇指指间（IP）关节或其他手指近端指间（PIP）关节的挛缩相关联。对177个患指进行了轴向图像的超声测量，其中包括17个拇指和34个被判定存在IP或PIP关节挛缩的其他手指，以及77个对侧对照手指。挛缩组的A1滑车在所有手指中均显著厚于非挛缩组，而屈肌腱仅在拇指以外的手指中更厚。在使用计算出的临界值进行的分析中，拇指的A1滑车增厚以及其他手指的A1滑车和屈肌腱增厚与IP或PIP关节挛缩具有统计学上的显著相关性。本研究通过超声证实了先前的报道，即屈肌腱增粗有助于PIP关节挛缩的发病机制。
