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Modifying vascular calcification in diabetes mellitus: contribution of O-GlcNAcylation.

作者信息

Rogers Maximillian A, Aikawa Elena

机构信息

From the Center for Interdisciplinary Cardiovascular Sciences (M.A.R., E.A.) and Center of Excellence in Vascular Biology (E.A.), Division of Cardiovascular Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA.

出版信息

Circ Res. 2014 Mar 28;114(7):1074-6. doi: 10.1161/CIRCRESAHA.114.303684.

DOI:10.1161/CIRCRESAHA.114.303684
PMID:24677232
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3989539/
Abstract
摘要

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本文引用的文献

1
Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus.O-连接的 N-乙酰葡萄糖胺对 AKT 的激活诱导糖尿病中的血管钙化。
Circ Res. 2014 Mar 28;114(7):1094-102. doi: 10.1161/CIRCRESAHA.114.302968. Epub 2014 Feb 13.
2
Macrophage-derived matrix vesicles: an alternative novel mechanism for microcalcification in atherosclerotic plaques.巨噬细胞衍生的基质小泡：动脉粥样硬化斑块中微钙化的另一种新机制。
Circ Res. 2013 Jun 21;113(1):72-7. doi: 10.1161/CIRCRESAHA.113.301036. Epub 2013 Apr 24.
3
Vascular calcification: an update on mechanisms and challenges in treatment.血管钙化：机制和治疗挑战的最新进展。
Calcif Tissue Int. 2013 Oct;93(4):365-73. doi: 10.1007/s00223-013-9712-z. Epub 2013 Mar 1.
4
Identification of O-linked N-acetylglucosamine (O-GlcNAc)-modified osteoblast proteins by electron transfer dissociation tandem mass spectrometry reveals proteins critical for bone formation.通过电子转移解离串联质谱鉴定 O-连接 N-乙酰葡萄糖胺（O-GlcNAc）修饰的成骨细胞蛋白，揭示了对骨形成至关重要的蛋白。
Mol Cell Proteomics. 2013 Apr;12(4):945-55. doi: 10.1074/mcp.M112.026633. Epub 2013 Feb 26.
5
The increase in O-linked N-acetylglucosamine protein modification stimulates chondrogenic differentiation both in vitro and in vivo.O-连接 N-乙酰葡萄糖胺蛋白修饰的增加可刺激体外和体内的软骨分化。
J Biol Chem. 2012 Sep 28;287(40):33615-28. doi: 10.1074/jbc.M112.354241. Epub 2012 Aug 2.
6
Molecular imaging insights into early inflammatory stages of arterial and aortic valve calcification.分子影像学对动脉和主动脉瓣钙化的早期炎症阶段的研究进展。
Circ Res. 2011 May 27;108(11):1381-91. doi: 10.1161/CIRCRESAHA.110.234146.
7
Calcium regulates key components of vascular smooth muscle cell-derived matrix vesicles to enhance mineralization.钙调节血管平滑肌细胞来源的基质小泡的关键成分，以增强矿化。
Circ Res. 2011 Jun 24;109(1):e1-12. doi: 10.1161/CIRCRESAHA.110.238808. Epub 2011 May 12.
8
Cross talk between O-GlcNAcylation and phosphorylation: roles in signaling, transcription, and chronic disease.O-糖基化与磷酸化的对话：在信号转导、转录和慢性疾病中的作用。
Annu Rev Biochem. 2011;80:825-58. doi: 10.1146/annurev-biochem-060608-102511.
9
O-GlcNAc modification, insulin signaling and diabetic complications.O-连接的 N-乙酰氨基葡萄糖修饰、胰岛素信号转导与糖尿病并发症。
Diabetes Metab. 2010 Dec;36(6 Pt 1):423-35. doi: 10.1016/j.diabet.2010.09.001. Epub 2010 Nov 11.
10
Vascular calcification: pathobiology of a multifaceted disease.血管钙化：一种多方面疾病的病理生物学
Circulation. 2008 Jun 3;117(22):2938-48. doi: 10.1161/CIRCULATIONAHA.107.743161.