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Pyk2 contributes to reepithelialization by promoting MMP expression. Focus on "Delayed skin wound repair in proline-rich protein tyrosine kinase 2 knockout mice".Pyk2通过促进基质金属蛋白酶（MMP）表达来促进上皮再形成。聚焦于“富含脯氨酸的蛋白酪氨酸激酶2基因敲除小鼠的皮肤伤口修复延迟”。
Am J Physiol Cell Physiol. 2014 May 15;306(10):C887-8. doi: 10.1152/ajpcell.00098.2014. Epub 2014 Apr 2.
2
Delayed skin wound repair in proline-rich protein tyrosine kinase 2 knockout mice.脯氨酸丰富蛋白酪氨酸激酶 2 基因敲除小鼠皮肤伤口愈合延迟。
Am J Physiol Cell Physiol. 2014 May 15;306(10):C899-909. doi: 10.1152/ajpcell.00331.2013. Epub 2014 Mar 5.
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J Invest Dermatol. 2007 May;127(5):1094-106. doi: 10.1038/sj.jid.5700662. Epub 2007 Jan 4.
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Transcriptional control of skin reepithelialization.皮肤再上皮化的转录控制。
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Pyk2 is essential for astrocytes mobility following brain lesion.Pyk2 对于脑损伤后星形胶质细胞的迁移是必需的。
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CCN1 accelerates re-epithelialization by promoting keratinocyte migration and proliferation during cutaneous wound healing.CCN1 通过促进角质形成细胞迁移和增殖加速皮肤伤口愈合的再上皮化。
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A Novel Synthetic Material, BMM, Accelerates Wound Repair by Stimulating Re-Epithelialization and Fibroblast Activation.一种新型合成材料 BMM 通过刺激再上皮化和纤维母细胞活化来加速伤口修复。
Int J Mol Sci. 2018 Apr 11;19(4):1164. doi: 10.3390/ijms19041164.

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FOXO1 deletion in keratinocytes improves diabetic wound healing through MMP9 regulation.角质细胞中 FOXO1 的缺失通过调节 MMP9 来改善糖尿病伤口愈合。
Sci Rep. 2017 Sep 5;7(1):10565. doi: 10.1038/s41598-017-10999-3.
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Targeting focal adhesion kinase renders pancreatic cancers responsive to checkpoint immunotherapy.靶向粘着斑激酶可使胰腺癌对检查点免疫疗法产生反应。
Nat Med. 2016 Aug;22(8):851-60. doi: 10.1038/nm.4123. Epub 2016 Jul 4.
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FOXO1, TGF-β regulation and wound healing.叉头框蛋白O1、转化生长因子-β调控与伤口愈合
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本文引用的文献

1
Delayed skin wound repair in proline-rich protein tyrosine kinase 2 knockout mice.脯氨酸丰富蛋白酪氨酸激酶 2 基因敲除小鼠皮肤伤口愈合延迟。
Am J Physiol Cell Physiol. 2014 May 15;306(10):C899-909. doi: 10.1152/ajpcell.00331.2013. Epub 2014 Mar 5.
2
Downregulation of CD9 in keratinocyte contributes to cell migration via upregulation of matrix metalloproteinase-9.角质细胞中 CD9 的下调通过上调基质金属蛋白酶-9 促进细胞迁移。
PLoS One. 2013 Oct 16;8(10):e77806. doi: 10.1371/journal.pone.0077806. eCollection 2013.
3
FOXO1 promotes wound healing through the up-regulation of TGF-β1 and prevention of oxidative stress.FOXO1 通过上调 TGF-β1 和预防氧化应激促进伤口愈合。
J Cell Biol. 2013 Oct 28;203(2):327-43. doi: 10.1083/jcb.201305074. Epub 2013 Oct 21.
4
A review of the influence of growth factors and cytokines in in vitro human keratinocyte migration.生长因子和细胞因子对体外培养人角质形成细胞迁移影响的综述
Cytokine. 2013 Apr;62(1):1-21. doi: 10.1016/j.cyto.2013.02.015. Epub 2013 Mar 11.
5
Nod2 deficiency impairs inflammatory and epithelial aspects of the cutaneous wound-healing response.Nod2 缺乏损害皮肤创伤愈合反应的炎症和上皮方面。
J Pathol. 2013 Jan;229(1):121-31. doi: 10.1002/path.4095.
6
Matrix metalloproteinases and epidermal wound repair.基质金属蛋白酶与表皮创伤修复。
Cell Tissue Res. 2013 Feb;351(2):255-68. doi: 10.1007/s00441-012-1410-z. Epub 2012 Apr 18.
7
Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis.缺乏基质金属蛋白酶-9的小鼠表现出伤口愈合延迟，这与上皮再形成延迟和胶原纤维形成紊乱有关。
Matrix Biol. 2009 Mar;28(2):65-73. doi: 10.1016/j.matbio.2009.01.001. Epub 2009 Jan 20.
8
alpha v beta3 and alpha5beta1 integrin recycling pathways dictate downstream Rho kinase signaling to regulate persistent cell migration.αvβ3和α5β1整合素循环途径决定下游Rho激酶信号传导，以调节细胞持续迁移。
J Cell Biol. 2007 May 7;177(3):515-25. doi: 10.1083/jcb.200609004.
9
Overexpression of TIMP-1 under the MMP-9 promoter interferes with wound healing in transgenic mice.在基质金属蛋白酶-9（MMP-9）启动子调控下组织金属蛋白酶抑制剂-1（TIMP-1）的过表达会干扰转基因小鼠的伤口愈合。
Cell Tissue Res. 2004 Jan;315(1):27-37. doi: 10.1007/s00441-003-0814-1. Epub 2003 Oct 21.

Pyk2 contributes to reepithelialization by promoting MMP expression. Focus on "Delayed skin wound repair in proline-rich protein tyrosine kinase 2 knockout mice".

作者信息

Graves Dana T, Wu Yingying, Badadani Mallikarjun

机构信息

School of Dental Medicine, University of Pennsylvania, Philadelphia, Pennsylvania; and

School of Dental Medicine, University of Pennsylvania, Philadelphia, Pennsylvania; and State Key Laboratory of Oral Disease, Sichuan University, Sichuan, People's Republic of China.

出版信息

Am J Physiol Cell Physiol. 2014 May 15;306(10):C887-8. doi: 10.1152/ajpcell.00098.2014. Epub 2014 Apr 2.

DOI:10.1152/ajpcell.00098.2014
PMID:24696142
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4024714/
Abstract
摘要