右旋糖酐对红细胞的保护作用，使其免受低pH诱导的溶血。
Dextran protection of erythrocytes from low-pH-induced hemolysis.
作者信息
Cudd A, Arvinte T, Schulz B, Nicolau C
机构信息
Biophor Corporation, Texas A & M University Research Park, College Station 77840.
出版信息
FEBS Lett. 1989 Jul 3;250(2):293-6. doi: 10.1016/0014-5793(89)80741-0.
Low-pH-induced hemolysis of erythrocytes is inhibited by dextrans. The protective effect was observed with dextrans larger than 40 kDa. Electron microscopy showed dextrans of 150 kDa in a tight association with the erythrocyte membrane. These results indicate that dextrans stop the low-pH-induced hemolysis by interacting with the acid-induced defects in the erythrocyte membrane [(1989) Biochim. Biophys. Acta, in press].
低pH诱导的红细胞溶血受到右旋糖酐的抑制。分子量大于40 kDa的右旋糖酐具有这种保护作用。电子显微镜显示150 kDa的右旋糖酐与红细胞膜紧密结合。这些结果表明，右旋糖酐通过与红细胞膜上酸诱导的缺陷相互作用来阻止低pH诱导的溶血[（1989年）《生物化学与生物物理学报》，即将发表]。
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