Comment on "Killer Ig-like receptor 2DL4 does not mediate NK Cell IFN-γ responses to soluble HLA-G preparations".
作者信息
Rajagopalan Sumati, Long Eric O
机构信息
Laboratory of Immunogenetics, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Rockville, MD 20852.
出版信息
J Immunol. 2014 May 1;192(9):4003. doi: 10.4049/jimmunol.1400430.
Abstract
摘要
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Comment on "Killer Ig-like receptor 2DL4 does not mediate NK Cell IFN-γ responses to soluble HLA-G preparations".对“杀伤细胞免疫球蛋白样受体2DL4不介导自然杀伤细胞对可溶性HLA - G制剂的γ干扰素反应”的评论
J Immunol. 2014 May 1;192(9):4003. doi: 10.4049/jimmunol.1400430.
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本文引用的文献
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Killer Ig-like receptor 2DL4 does not mediate NK cell IFN-γ responses to soluble HLA-G preparations.杀伤细胞免疫球蛋白样受体 2DL4 不能介导 NK 细胞对可溶性 HLA-G 制剂的 IFN-γ 反应。
J Immunol. 2014 Jan 15;192(2):732-40. doi: 10.4049/jimmunol.1301748. Epub 2013 Dec 11.
2
Cellular senescence induced by CD158d reprograms natural killer cells to promote vascular remodeling.CD158d 诱导的细胞衰老重编程自然杀伤细胞以促进血管重塑。
Proc Natl Acad Sci U S A. 2012 Dec 11;109(50):20596-601. doi: 10.1073/pnas.1208248109. Epub 2012 Nov 26.
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DNA-PKcs controls an endosomal signaling pathway for a proinflammatory response by natural killer cells.DNA-PKcs 通过自然杀伤细胞控制内体信号通路促进炎症反应。
Sci Signal. 2010 Feb 23;3(110):ra14. doi: 10.1126/scisignal.2000467.
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Activation of NK cells by an endocytosed receptor for soluble HLA-G.可溶性 HLA - G 的内吞受体对自然杀伤细胞的激活作用。
PLoS Biol. 2006 Jan;4(1):e9. doi: 10.1371/journal.pbio.0040009.
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