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平滑肌细胞条件培养基可提高牛肺动脉内皮细胞的血管紧张素转换酶水平。

Smooth muscle cell conditioned medium elevates angiotensin-converting enzyme of bovine pulmonary artery endothelial cells.

作者信息

Yu F S, Lee S L, Fanburg B L

机构信息

Department of Medicine, New England Medical Center Hospital, Boston, MA 02111.

出版信息

Am J Respir Cell Mol Biol. 1989 Nov;1(5):401-5. doi: 10.1165/ajrcmb/1.5.401.

Abstract

Conditioned medium obtained from bovine pulmonary artery smooth muscle cells (SMC) in culture was found to elevate angiotensin-I-converting enzyme (ACE) of bovine pulmonary artery endothelial cells (EC) by 2- to 3-fold upon incubation for 24 to 48 h. The elevation in ACE was time dependent and inhibited by 10(-6) M cycloheximide, making it likely that the elevation was related to new protein synthesis by EC. Conditioned medium from EC failed to produce the same effect. The stimulatory effect of SMC conditioned medium on EC ACE was uninfluenced by exposures to anoxia as compared with room air, either during conditioning of medium or while assaying the effect of conditioned medium on EC ACE. Inhibitors of prostaglandin metabolism and calcium transport failed to influence the stimulatory effect of SMC conditioned medium on EC ACE. The stimulatory effect on EC ACE by conditioned medium was additive to that produced by Ca2+ ionophore A23187 and 3-isobutyl-1-methylxanthine and, similar to baseline ACE, was inhibited by 3 x 10(-8) M ouabain. Thus, SMC produce a factor that regulates the level of EC ACE.

摘要

研究发现,培养的牛肺动脉平滑肌细胞(SMC)产生的条件培养基在与牛肺动脉内皮细胞(EC)共同孵育24至48小时后,可使EC的血管紧张素I转换酶(ACE)升高2至3倍。ACE的升高呈时间依赖性,并被10⁻⁶ M的环己酰亚胺抑制,这表明该升高可能与EC的新蛋白合成有关。EC的条件培养基未能产生相同的效果。与在空气中培养相比,无论是在培养基条件培养期间还是在检测条件培养基对EC ACE的作用时,SMC条件培养基对EC ACE的刺激作用不受缺氧暴露的影响。前列腺素代谢抑制剂和钙转运抑制剂均未能影响SMC条件培养基对EC ACE的刺激作用。条件培养基对EC ACE的刺激作用与Ca²⁺离子载体A23187和3 - 异丁基 - 1 - 甲基黄嘌呤产生的刺激作用具有相加性,并且与基线ACE相似,被3×10⁻⁸ M的哇巴因抑制。因此,SMC产生一种调节EC ACE水平的因子。

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