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Response to letter regarding article, "dysregulation of hydrogen sulfide (H2S) producing enzyme cystathionine γ-lyase (CSE) contributes to maternal hypertension and placental abnormalities in preeclampsia".

作者信息

Wang Keqing, Ahmad Shakil, Cai Meng, Rennie Jillian, Fujisawa Takeshi, Crispi Fatima, Baily James, Miller Mark R, Cudmore Melissa, Hadoke Patrick W F, Wang Rui, Gratacós Eduard, Buhimschi Irina A, Buhimschi Catalin S, Ahmed Asif

机构信息

Vascular Medicine Unit, Aston Medical School, Aston University, Birmingham, UK.

Gustav Born Centre for Vascular Biology and BHF Centre for Cardiovascular Sciences, University of Edinburgh, Edinburgh, UK.

出版信息

Circulation. 2014 Jun 3;129(22):e517-8. doi: 10.1161/CIRCULATIONAHA.114.010273.

DOI:10.1161/CIRCULATIONAHA.114.010273
PMID:24891630
Abstract
摘要

相似文献

1
Response to letter regarding article, "dysregulation of hydrogen sulfide (H2S) producing enzyme cystathionine γ-lyase (CSE) contributes to maternal hypertension and placental abnormalities in preeclampsia".对关于文章《产硫化氢（H2S）的酶胱硫醚γ-裂解酶（CSE）失调导致子痫前期孕妇高血压和胎盘异常》的来信的回复
Circulation. 2014 Jun 3;129(22):e517-8. doi: 10.1161/CIRCULATIONAHA.114.010273.
2
Letter by Tsikas and Cooper regarding article, "dysregulation of hydrogen sulfide (H2S) producing enzyme cystathionine γ-lyase (CSE) contributes to maternal hypertension and placental abnormalities in preeclampsia".齐卡斯和库珀就文章《产硫化氢（H2S）的酶胱硫醚γ-裂解酶（CSE）失调导致子痫前期孕妇高血压和胎盘异常》所写的信。
Circulation. 2014 Jun 3;129(22):e516. doi: 10.1161/CIRCULATIONAHA.113.004320.
3
Dysregulation of hydrogen sulfide producing enzyme cystathionine γ-lyase contributes to maternal hypertension and placental abnormalities in preeclampsia.胱硫醚 γ-裂解酶产生酶的失调导致子痫前期的母体高血压和胎盘异常。
Circulation. 2013 Jun 25;127(25):2514-22. doi: 10.1161/CIRCULATIONAHA.113.001631. Epub 2013 May 23.
4
Sulfane sustains vascular health: insights into cystathionine γ-lyase function.硫化氢维持血管健康：对胱硫醚γ-裂解酶功能的见解。
Circulation. 2013 Jun 25;127(25):2472-4. doi: 10.1161/CIRCULATIONAHA.113.003489. Epub 2013 May 23.
5
CD4 T-Cell Endogenous Cystathionine γ Lyase-Hydrogen Sulfide Attenuates Hypertension by Sulfhydrating Liver Kinase B1 to Promote T Regulatory Cell Differentiation and Proliferation.CD4 T 细胞内源性半胱氨酸 γ 裂解酶-硫化氢通过巯基化肝激酶 B1 促进调节性 T 细胞分化和增殖来减轻高血压。
Circulation. 2020 Nov 3;142(18):1752-1769. doi: 10.1161/CIRCULATIONAHA.119.045344. Epub 2020 Sep 9.
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Dysregulation of cystathionine γ-lyase (CSE)/hydrogen sulfide pathway contributes to ox-LDL-induced inflammation in macrophage.胱硫醚γ-裂解酶（CSE）/硫化氢通路的失调导致巨噬细胞中氧化型 LDL 诱导的炎症反应。
Cell Signal. 2013 Nov;25(11):2255-62. doi: 10.1016/j.cellsig.2013.07.010. Epub 2013 Jul 18.
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Hydrogen sulfide producing enzymes in pregnancy and preeclampsia.妊娠和子痫前期中产生硫化氢的酶。
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8
MiR 20a,-20b and -200c are involved in hydrogen sulfide stimulation of VEGF production in human placental trophoblasts.微小RNA 20a、-20b和-200c参与硫化氢对人胎盘滋养层细胞中血管内皮生长因子产生的刺激作用。
Placenta. 2016 Mar;39:101-10. doi: 10.1016/j.placenta.2016.01.019. Epub 2016 Jan 22.
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-3-Carboxypropyl-l-cysteine specifically inhibits cystathionine γ-lyase-dependent hydrogen sulfide synthesis.-3-羧丙基-L-半胱氨酸特异性抑制胱硫醚 γ-裂解酶依赖的硫化氢合成。
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MiR133b is involved in endogenous hydrogen sulfide suppression of sFlt-1 production in human placenta.微小RNA133b参与内源性硫化氢对人胎盘可溶性血管内皮生长因子受体1产生的抑制作用。
Placenta. 2017 Apr;52:33-40. doi: 10.1016/j.placenta.2017.02.012. Epub 2017 Feb 12.

引用本文的文献

1
Hydrogen Sulfide Attenuated Angiotensin II-Induced Sympathetic Excitation in Offspring of Renovascular Hypertensive Rats.硫化氢减轻肾血管性高血压大鼠子代中血管紧张素II诱导的交感神经兴奋
Front Pharmacol. 2020 Sep 11;11:565726. doi: 10.3389/fphar.2020.565726. eCollection 2020.
2
Parental Renovascular Hypertension-Induced Autonomic Dysfunction in Male Offspring Is Improved by Prenatal or Postnatal Treatment With Hydrogen Sulfide.产前或产后给予硫化氢治疗可改善父源性肾血管性高血压诱导的雄性子代自主神经功能障碍。
Front Physiol. 2019 Sep 19;10:1184. doi: 10.3389/fphys.2019.01184. eCollection 2019.
3
Pre-eclampsia: its pathogenesis and pathophysiolgy.
子痫前期：其发病机制与病理生理学
Cardiovasc J Afr. 2016 Mar-Apr;27(2):71-8. doi: 10.5830/CVJA-2016-009.