Yuasa Sho, Sato Masahito, Kitazawa Hitoshi, Okabe Masaaki, Komatsu Yasushi, Koshikawa Tomoyasu, Miyajima Seiichi, Aizawa Yoshifusa
Tachikawa General Hospital, Cardiology, Nagaoka, Japan.
Miyajima Clinics, Tsubame, Japan.
J Electrocardiol. 2014 Sep-Oct;47(5):721-4. doi: 10.1016/j.jelectrocard.2014.04.022. Epub 2014 May 4.
The patient was a 33-year-old male. Twenty years ago, he underwent radiofrequency catheter ablation for idiopathic sustained monomorphic ventricular tachycardia (VT) with an RBBB and superior axis pattern. The VT was inducible by programmed stimulation and entrained by rapid pacing. At this presentation, he developed palpitation and VT with the same morphology at the peak exercise on a treadmill with appearance of typical ECG pattern for Brugada syndrome (BrS). Pilsicainide induced the typical ECG pattern and premature ventricular beats (PVBs) of the same morphology as VT. The relationship between BrS and VT of left ventricular origin was discussed.
该患者为一名33岁男性。20年前，他因特发性持续性单形性室性心动过速（VT）伴右束支传导阻滞和电轴上偏模式接受了射频导管消融术。该室性心动过速可通过程序刺激诱发，并可被快速起搏拖带。此次就诊时，他在跑步机上进行峰值运动时出现心悸和形态相同的室性心动过速，伴有Brugada综合征（BrS）的典型心电图表现。吡西卡尼诱发了典型的心电图表现以及与室性心动过速形态相同的室性早搏（PVBs）。讨论了Brugada综合征与左心室起源室性心动过速之间的关系。
