MDM2 SNP309 and risk of endometrial cancer.

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作者信息

Knappskog Stian, Lønning Per Eystein

机构信息

Section of Oncology, Department of Clinical Science, University of Bergen, Bergen, Norway,

出版信息

Tumour Biol. 2014 Aug;35(8):7285-6. doi: 10.1007/s13277-014-2244-y. Epub 2014 Jun 26.

DOI:10.1007/s13277-014-2244-y

PMID:24964960

Abstract

摘要

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MDM2 SNP309 and risk of endometrial cancer.

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Population distribution and ancestry of the cancer protective MDM2 SNP285 (rs117039649).癌症保护性MDM2 SNP285（rs117039649）的人群分布及祖先情况

Oncotarget. 2014 Sep 30;5(18):8223-34. doi: 10.18632/oncotarget.1910.

Common variant on MDM2 contributes to endometrial cancer susceptibility: evidence based on 7 studies.MDM2基因的常见变异与子宫内膜癌易感性相关：基于7项研究的证据

Tumour Biol. 2014 Aug;35(8):7555-60. doi: 10.1007/s13277-014-1886-0. Epub 2014 May 3.

MDM2 SNP309 polymorphism contributes to endometrial cancer susceptibility: evidence from a meta-analysis.MDM2基因SNP309多态性与子宫内膜癌易感性相关：一项荟萃分析的证据

J Exp Clin Cancer Res. 2013 Nov 3;32(1):85. doi: 10.1186/1756-9966-32-85.

DDX3 loss by p53 inactivation promotes tumor malignancy via the MDM2/Slug/E-cadherin pathway and poor patient outcome in non-small-cell lung cancer.p53 失活导致 DDX3 缺失通过 MDM2/Slug/E-钙黏蛋白通路促进肿瘤恶性进展并导致非小细胞肺癌患者预后不良。

Oncogene. 2014 Mar 20;33(12):1515-26. doi: 10.1038/onc.2013.107. Epub 2013 Apr 15.

SNP285C modulates oestrogen receptor/Sp1 binding to the MDM2 promoter and reduces the risk of endometrial but not prostatic cancer.SNP285C 调节雌激素受体/Sp1 与 MDM2 启动子的结合，降低子宫内膜癌但不增加前列腺癌的风险。

Eur J Cancer. 2012 Sep;48(13):1988-96. doi: 10.1016/j.ejca.2011.10.024. Epub 2011 Nov 24.

MDM2 promoter SNP285 and SNP309; phylogeny and impact on cancer risk.MDM2基因启动子SNP285和SNP309；系统发育及其对癌症风险的影响。

Oncotarget. 2011 Mar;2(3):251-8. doi: 10.18632/oncotarget.243.

The MDM2 promoter SNP285C/309G haplotype diminishes Sp1 transcription factor binding and reduces risk for breast and ovarian cancer in Caucasians.MDM2 启动子 SNP285C/309G 单倍型降低了 Sp1 转录因子的结合，并降低了白种人患乳腺癌和卵巢癌的风险。

Cancer Cell. 2011 Feb 15;19(2):273-82. doi: 10.1016/j.ccr.2010.12.019.

A single nucleotide polymorphism in the MDM2 promoter attenuates the p53 tumor suppressor pathway and accelerates tumor formation in humans.MDM2启动子中的单核苷酸多态性会减弱p53肿瘤抑制通路，并加速人类肿瘤的形成。

Cell. 2004 Nov 24;119(5):591-602. doi: 10.1016/j.cell.2004.11.022.

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