Su I-Chang, Cruz Juan Pablo, Krings Timo
Division of Neuroradiology, Department of Medical Imaging, Toronto Western Hospital, University Health Network; Toronto, Ontario, Canada - Division of Neurosurgery, Department of Surgery, Taipei Cathay General Hospital; Taipei, Taiwan -
Division of Neuroradiology, Department of Medical Imaging, Toronto Western Hospital, University Health Network; Toronto, Ontario, Canada.
Interv Neuroradiol. 2014 Jul-Aug;20(4):476-81. doi: 10.15274/INR-2014-10057. Epub 2014 Aug 28.
Direct carotid-cavernous fistulas (CCFs) secondary to a ruptured intracavernous carotid aneurysm (ICCA) are usually high-flow lesions. On very rare occasions, a ruptured ICCA may present as a low-flow CCF, which poses a diagnostic and therapeutic dilemma whether the aneurysm and the observed fistula are causally related. Herein, we describe a rare case in which a ruptured ICCA resulted in a low-flow CCF. We demonstrated our approach to clarify this clinical scenario, and also propose a possible pathomechanism to explain the existence of low-flow direct CCF.
海绵窦内颈动脉动脉瘤(ICCA)破裂继发的直接型颈内动脉海绵窦瘘(CCF)通常为高流量病变。在极少数情况下,破裂的ICCA可能表现为低流量CCF,这就产生了一个诊断和治疗难题,即动脉瘤与观察到的瘘是否存在因果关系。在此,我们描述了一例罕见病例,即破裂的ICCA导致了低流量CCF。我们展示了我们澄清这一临床情况的方法,并提出了一种可能的发病机制来解释低流量直接型CCF的存在。