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Identification and characterization of a p53 gene mutation in a human osteosarcoma cell line.

作者信息

Romano J W, Ehrhart J C, Duthu A, Kim C M, Appella E, May P

机构信息

Laboratory of Cell Biology, National Cancer Institute, NIH, Bethesda, Maryland 20892.

出版信息

Oncogene. 1989 Dec;4(12):1483-8.

PMID:2531855
Abstract

The nuclear phosphoprotein p53 occurs at elevated levels in many transformed cells. Mutant forms of mouse p53 possess enhanced transforming activity compared with wild type p53. Mutant mouse p53 proteins form complexes with the 70 kDa family of heat shock proteins (HSPs). We previously demonstrated an association between p53 and the 70 kDa HSPs in the human osteosarcoma (HOS) derived cell line HOS-SL. We report here the molecular cloning and sequencing of the p53 gene from HOS-SL cells, and demonstrate that it is in fact mutant. Further, analysis of similar HOS-derived cell lines demonstrates that they also encode the same mutant form of p53, whereas the wild type form of p53 appears to be lost in these cells. Stability studies demonstrate an increased half life of the p53 protein in these cells, in keeping with its association with the HSP 70 proteins. A potential role for this p53 mutant in the transformation process is discussed.

摘要

相似文献

1
Identification and characterization of a p53 gene mutation in a human osteosarcoma cell line.
Oncogene. 1989 Dec;4(12):1483-8.
2
Specific interaction between a subset of the p53 protein family and heat shock proteins hsp72/hsc73 in a human osteosarcoma cell line.
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Mutations in the p53 gene occur in diverse human tumour types.p53基因的突变发生在多种人类肿瘤类型中。
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