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Endotoxin and AKI: Macrophages Protect after Preconditioning.内毒素与急性肾损伤：预处理后巨噬细胞发挥保护作用。
J Am Soc Nephrol. 2015 Jun;26(6):1231-2. doi: 10.1681/ASN.2014101042. Epub 2014 Nov 14.
2
The macrophage mediates the renoprotective effects of endotoxin preconditioning.巨噬细胞介导内毒素预处理的肾脏保护作用。
J Am Soc Nephrol. 2015 Jun;26(6):1347-62. doi: 10.1681/ASN.2014060561. Epub 2014 Nov 14.
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p66SHC-mediated mitochondrial dysfunction in renal proximal tubule cells during oxidative injury.p66SHC 介导的氧化损伤过程中肾近端小管细胞线粒体功能障碍。
Am J Physiol Renal Physiol. 2010 May;298(5):F1214-21. doi: 10.1152/ajprenal.00639.2009. Epub 2010 Jan 6.
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Remote ischemic preconditioning for kidney protection: GSK3β-centric insights into the mechanism of action.远程缺血预处理对肾脏的保护作用：以糖原合成酶激酶3β为核心对作用机制的深入了解
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Unique proximal tubular cell injury and the development of acute kidney injury in adult patients with minimal change nephrotic syndrome.微小病变型肾病综合征成年患者独特的近端肾小管细胞损伤与急性肾损伤的发生
BMC Nephrol. 2017 Nov 28;18(1):339. doi: 10.1186/s12882-017-0756-6.
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Hyperoxia-induced preconditioning against renal ischemic injury is mediated by reactive oxygen species but not related to heat shock proteins 70 and 32.高氧诱导的肾缺血损伤预处理由活性氧介导，但与热休克蛋白70和32无关。
Surgery. 2015 Jun;157(6):1014-22. doi: 10.1016/j.surg.2015.01.025. Epub 2015 Apr 3.
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Altered cholesterol localization and caveolin expression during the evolution of acute renal failure.急性肾衰竭进展过程中胆固醇定位及小窝蛋白表达的改变
Kidney Int. 2002 May;61(5):1674-83. doi: 10.1046/j.1523-1755.2002.00316.x.
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Diglycolic acid inhibits succinate dehydrogenase activity in human proximal tubule cells leading to mitochondrial dysfunction and cell death.二甘醇酸抑制人近端肾小管细胞琥珀酸脱氢酶活性，导致线粒体功能障碍和细胞死亡。
Toxicol Lett. 2013 Aug 29;221(3):176-84. doi: 10.1016/j.toxlet.2013.06.231. Epub 2013 Jul 1.
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Remote ischemic preconditioning has a neutral effect on the incidence of kidney injury after coronary artery bypass graft surgery.远程缺血预处理对冠状动脉旁路移植术后肾损伤的发生率无影响。
Kidney Int. 2015 Feb;87(2):473-81. doi: 10.1038/ki.2014.259. Epub 2014 Jul 30.
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p66shc and gender-specific dimorphism in acute renal injury.p66shc与急性肾损伤中的性别特异性二态性
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Tetramethylpyrazine: An Active Ingredient of Chinese Herbal Medicine With Therapeutic Potential in Acute Kidney Injury and Renal Fibrosis.川芎嗪：一种具有治疗急性肾损伤和肾纤维化潜力的中草药活性成分。
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Exosome-mediated pyroptosis of miR-93-TXNIP-NLRP3 leads to functional difference between M1 and M2 macrophages in sepsis-induced acute kidney injury.外泌体介导的 miR-93-TXNIP-NLRP3 细胞焦亡导致脓毒症诱导的急性肾损伤中 M1 和 M2 巨噬细胞功能差异。
J Cell Mol Med. 2021 May;25(10):4786-4799. doi: 10.1111/jcmm.16449. Epub 2021 Mar 21.
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Dexmedetomidine protects against acute kidney injury through downregulating inflammatory reactions in endotoxemia rats.右美托咪定通过下调内毒素血症大鼠的炎症反应来预防急性肾损伤。
Biomed Rep. 2015 May;3(3):365-370. doi: 10.3892/br.2015.427. Epub 2015 Feb 12.

本文引用的文献

1
The macrophage mediates the renoprotective effects of endotoxin preconditioning.巨噬细胞介导内毒素预处理的肾脏保护作用。
J Am Soc Nephrol. 2015 Jun;26(6):1347-62. doi: 10.1681/ASN.2014060561. Epub 2014 Nov 14.
2
Anthracyclines induce DNA damage response-mediated protection against severe sepsis.蒽环类抗生素诱导 DNA 损伤反应介导的严重脓毒症保护作用。
Immunity. 2013 Nov 14;39(5):874-84. doi: 10.1016/j.immuni.2013.08.039. Epub 2013 Oct 31.
3
The immunobiology of toll-like receptor 4 agonists: from endotoxin tolerance to immunoadjuvants.Toll 样受体 4 激动剂的免疫生物学：从内毒素耐受到免疫佐剂。
Shock. 2013 Dec;40(6):451-62. doi: 10.1097/SHK.0000000000000042.
4
Disease tolerance as a defense strategy.疾病耐受力作为一种防御策略。
Science. 2012 Feb 24;335(6071):936-41. doi: 10.1126/science.1214935.
5
Endotoxin uptake by S1 proximal tubular segment causes oxidative stress in the downstream S2 segment.S1 近端肾小管段摄取内毒素导致下游 S2 段发生氧化应激。
J Am Soc Nephrol. 2011 Aug;22(8):1505-16. doi: 10.1681/ASN.2011020203. Epub 2011 Jul 22.
6
Sepsis and acute kidney injury.脓毒症与急性肾损伤。
J Am Soc Nephrol. 2011 Jun;22(6):999-1006. doi: 10.1681/ASN.2010050484. Epub 2011 May 12.
7
Bone marrow stromal cells attenuate sepsis via prostaglandin E(2)-dependent reprogramming of host macrophages to increase their interleukin-10 production.骨髓基质细胞通过前列腺素E(2)依赖的宿主巨噬细胞重编程来减轻脓毒症，从而增加其白细胞介素-10的产生。
Nat Med. 2009 Jan;15(1):42-9. doi: 10.1038/nm.1905. Epub 2008 Nov 21.
8
Lipopolysaccharide pretreatment protects from renal ischemia/reperfusion injury : possible connection to an interleukin-6-dependent pathway.脂多糖预处理可保护肾脏免受缺血/再灌注损伤：可能与白细胞介素-6依赖性途径有关。
Am J Pathol. 2000 Jan;156(1):287-93. doi: 10.1016/S0002-9440(10)64729-3.

Endotoxin and AKI: Macrophages Protect after Preconditioning.

作者信息

Yatim Karim M, Oberbarnscheidt Martin H

机构信息

Thomas E. Starzl Transplantation Institute, University of Pittsburgh, Pittsburgh, Pennsylvania.

Thomas E. Starzl Transplantation Institute, University of Pittsburgh, Pittsburgh, Pennsylvania

出版信息

J Am Soc Nephrol. 2015 Jun;26(6):1231-2. doi: 10.1681/ASN.2014101042. Epub 2014 Nov 14.

DOI:10.1681/ASN.2014101042
PMID:25398786
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4446886/
Abstract
摘要