Endotoxin and AKI: Macrophages Protect after Preconditioning.
作者信息
Yatim Karim M, Oberbarnscheidt Martin H
机构信息
Thomas E. Starzl Transplantation Institute, University of Pittsburgh, Pittsburgh, Pennsylvania.
Thomas E. Starzl Transplantation Institute, University of Pittsburgh, Pittsburgh, Pennsylvania
出版信息
J Am Soc Nephrol. 2015 Jun;26(6):1231-2. doi: 10.1681/ASN.2014101042. Epub 2014 Nov 14.
Abstract
摘要
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本文引用的文献
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J Am Soc Nephrol. 2015 Jun;26(6):1347-62. doi: 10.1681/ASN.2014060561. Epub 2014 Nov 14.
2
Anthracyclines induce DNA damage response-mediated protection against severe sepsis.蒽环类抗生素诱导 DNA 损伤反应介导的严重脓毒症保护作用。
Immunity. 2013 Nov 14;39(5):874-84. doi: 10.1016/j.immuni.2013.08.039. Epub 2013 Oct 31.
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The immunobiology of toll-like receptor 4 agonists: from endotoxin tolerance to immunoadjuvants.Toll 样受体 4 激动剂的免疫生物学：从内毒素耐受到免疫佐剂。
Shock. 2013 Dec;40(6):451-62. doi: 10.1097/SHK.0000000000000042.
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Disease tolerance as a defense strategy.疾病耐受力作为一种防御策略。
Science. 2012 Feb 24;335(6071):936-41. doi: 10.1126/science.1214935.
5
Endotoxin uptake by S1 proximal tubular segment causes oxidative stress in the downstream S2 segment.S1 近端肾小管段摄取内毒素导致下游 S2 段发生氧化应激。
J Am Soc Nephrol. 2011 Aug;22(8):1505-16. doi: 10.1681/ASN.2011020203. Epub 2011 Jul 22.
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Sepsis and acute kidney injury.脓毒症与急性肾损伤。
J Am Soc Nephrol. 2011 Jun;22(6):999-1006. doi: 10.1681/ASN.2010050484. Epub 2011 May 12.
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Bone marrow stromal cells attenuate sepsis via prostaglandin E(2)-dependent reprogramming of host macrophages to increase their interleukin-10 production.骨髓基质细胞通过前列腺素E(2)依赖的宿主巨噬细胞重编程来减轻脓毒症，从而增加其白细胞介素-10的产生。
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