Abdalla Abuelmagd, Kelly Frank
Midland Regional Tullamore, Tullamore, Ireland.
BMJ Case Rep. 2014 Nov 20;2014:bcr2014206517. doi: 10.1136/bcr-2014-206517.
We present the case of a 65-year-old man with an atypical presentation of pulmonary embolism (PE) as ST elevation myocardial infarction (STEMI) with high troponin. He presented with acute exertional dyspnoea without chest pain. Since the initial ECG showed ST elevation anteroseptal (V1-V4) with concomitant deep Q waves, a delayed STEMI with probable left ventricular aneurysm was the working diagnosis and was treated accordingly. Nevertheless, his coronary angiography was normal and it was then that PE was suspected. D-dimer was found to be elevated and CT pulmonary angiography confirmed bilateral PE with a large thrombus within the right main pulmonary artery. The patient made good clinical recovery and his ST elevation resolved with anticoagulation. The source was found to be a deep vein thrombosis in his right leg. The treatment was not compromised by the delayed diagnosis as he received timely anticoagulation as part of STEMI management.
我们报告了一例65岁男性患者,其表现为非典型的肺栓塞(PE),呈现为伴有高肌钙蛋白的ST段抬高型心肌梗死(STEMI)。他因急性劳力性呼吸困难就诊,无胸痛症状。由于初始心电图显示前间壁(V1-V4)ST段抬高并伴有深Q波,初步诊断为延迟性STEMI伴可能的左心室室壁瘤,并据此进行了治疗。然而,他的冠状动脉造影结果正常,此时怀疑为PE。发现D-二聚体升高,CT肺动脉造影证实双侧PE,右主肺动脉内有一个大血栓。患者临床恢复良好,抗凝治疗后ST段抬高消失。栓子来源为其右腿的深静脉血栓形成。由于他在STEMI治疗过程中及时接受了抗凝治疗,延迟诊断并未影响治疗效果。