必需脂肪酸缺乏导致的肾小球固有巨噬细胞耗竭可预防肾小球肾炎。 - Suppr | 超能文献

Depletion of resident glomerular macrophages by essential fatty acid deficiency protects against glomerulonephritis.

作者信息

Lefkowith J B, Rovin B, Schreiner G F

机构信息

Dept. of Medicine, Washington University School of Medicine, St. Louis, Mo. 63110.

出版信息

Adv Prostaglandin Thromboxane Leukot Res. 1989;19:560-3.

Abstract

摘要

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Depletion of resident glomerular macrophages by essential fatty acid deficiency protects against glomerulonephritis.必需脂肪酸缺乏导致的肾小球固有巨噬细胞耗竭可预防肾小球肾炎。

Adv Prostaglandin Thromboxane Leukot Res. 1989;19:560-3.

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Leukotriene D4 (LTD4)/leukotriene B4 (LTB4) interactions in the pathophysiology of experimental glomerulonephritis.白三烯D4（LTD4）/白三烯B4（LTB4）在实验性肾小球肾炎病理生理学中的相互作用

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Essential fatty acid deficiency ameliorates acute renal dysfunction in the rat after the administration of the aminonucleoside of puromycin.在给予嘌呤霉素氨基核苷后，必需脂肪酸缺乏可改善大鼠的急性肾功能障碍。

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A relationship between proteinuria and acute tubulointerstitial disease in rats with experimental nephrotic syndrome.实验性肾病综合征大鼠蛋白尿与急性肾小管间质性疾病的关系。

Am J Pathol. 1991 May;138(5):1111-23.