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组蛋白去乙酰化酶抑制剂贝利司他联合阿扎胞苷治疗晚期髓系肿瘤的I期及药效学研究

A phase I and pharmacodynamic study of the histone deacetylase inhibitor belinostat plus azacitidine in advanced myeloid neoplasia.

作者信息

Odenike Olatoyosi, Halpern Anna, Godley Lucy A, Madzo Jozef, Karrison Theodore, Green Margaret, Fulton Noreen, Mattison Ryan J, Yee Karen W L, Bennett Meghan, Koval Gregory, Malnassy Gregory, Larson Richard A, Ratain Mark J, Stock Wendy

机构信息

Department of Medicine, The University of Chicago, Chicago, IL, USA,

出版信息

Invest New Drugs. 2015 Apr;33(2):371-9. doi: 10.1007/s10637-014-0194-2. Epub 2014 Dec 9.

Abstract

Background We hypothesized that targeting two mechanisms of epigenetic silencing would be additive or synergistic with regard to expression of specific target genes. The primary objective of the study was to establish the maximum tolerated dose (MTD) of belinostat in combination with a fixed dose of azacitidine (AZA). Methods In Part A of the study, patients received a fixed dose of AZA, with escalating doses of belinostat given on the same days 1-5, in a 28 day cycle. Part B was designed to evaluate the relative contribution of belinostat to the combination based on analysis of pharmacodynamic markers, and incorporated a design in which patients were randomized during cycle 1 to AZA alone, or the combination, at the maximally tolerated dose of belinostat. Results 56 patients with myeloid neoplasia were enrolled. Dose escalation was feasible in part A, up to 1000 mg/m(2) dose level of belinostat. In Part B, 18 patients were assessable for quantitative analysis of specific target genes. At day 5 of therapy, MDR1 was significantly up-regulated in the belinostat/AZA arm compared with AZA alone arm (p = 0.0023). There were 18 responses among the 56 patients. Conclusions The combination of belinostat and AZA is feasible and associated with clinical activity. The recommended phase II dose is 1000 mg/m(2) of belinostat plus 75 mg/m(2) of AZA on days 1-5, every 28 days. Upregulation in MDR1 was observed in the combination arm at day 5 compared with the AZA alone arm, suggesting a relative biologic contribution of belinostat to the combination.

摘要

背景 我们假设,针对两种表观遗传沉默机制对于特定靶基因的表达将具有累加或协同作用。本研究的主要目的是确定贝利司他与固定剂量阿扎胞苷(AZA)联合使用时的最大耐受剂量(MTD)。方法 在研究的A部分,患者接受固定剂量的AZA,并在第1 - 5天给予递增剂量的贝利司他,每28天为一个周期。B部分旨在通过对药效学标志物的分析评估贝利司他对联合用药的相对贡献,并采用一种设计,即患者在第1周期被随机分配至单独使用AZA或使用贝利司他最大耐受剂量的联合用药组。结果 56例骨髓增生异常患者入组。A部分剂量递增可行,贝利司他剂量最高达1000 mg/m²。在B部分,18例患者可用于特定靶基因的定量分析。治疗第5天时,与单独使用AZA组相比,贝利司他/AZA组的多药耐药蛋白1(MDR1)显著上调(p = 0.0023)。56例患者中有18例有反应。结论 贝利司他与AZA联合使用可行且具有临床活性。推荐的II期剂量为第1 - 5天,每28天一次,贝利司他1000 mg/m²加AZA 75 mg/m²。与单独使用AZA组相比,联合用药组在第5天时观察到MDR1上调,提示贝利司他对联合用药有相对生物学贡献。

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