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[高钾血症导致起搏器捕获和感知功能障碍:一例报告]

[Hyperkalemia-induced failure of pacemaker capture and sensing: a case report].

作者信息

Wang Y P, Chen B X, Su K J, Sun L J, Zhang Y, Guo L J, Gao W

机构信息

Department of Cardiology, Peking University Third Hospital, Beijing 100191, China.

出版信息

Beijing Da Xue Xue Bao Yi Xue Ban. 2014 Dec 18;46(6):980-2.

Abstract

Hyperkalemia may induce serious cardiac arrhythmia, with possible life-threatening effects. It may cause cardiac pacemaker (PMK) malfunctioning due to a reduction of the electronegativity of the resting myocardial potential. We report the case of a 71-year-old woman who had a previous history of chronic heart failure, chronic renal failure and DDI pacemaker. She was admitted for disturbance of consciousness. During hospitalization, she was observed for extreme hypotension, acute hyperkalemia, ventricular escape rhythm, associated with failure of pacemaker capture and sensing. She was treated with calcium chloride injection, followed by insulin/glucose and sodium bicarbonate infusions; the electrocardiogram recordings showed an correction of the PMK malfunctioning and serial improvement of the intraventricular conduction. This case supports that hyperkalemia should be closely monitored in the chronic heart failure patients combined with chronic renal failure.

摘要

高钾血症可能诱发严重的心律失常,具有危及生命的潜在影响。它可能因静息心肌电位的电负性降低而导致心脏起搏器(PMK)功能异常。我们报告一例71岁女性病例,她既往有慢性心力衰竭、慢性肾衰竭病史且植入了DDI起搏器。她因意识障碍入院。住院期间,观察到她出现极度低血压、急性高钾血症、室性逸搏心律,同时伴有起搏器捕获和感知功能障碍。给予氯化钙注射液治疗,随后输注胰岛素/葡萄糖和碳酸氢钠;心电图记录显示PMK功能异常得到纠正,室内传导逐渐改善。该病例支持对于合并慢性肾衰竭的慢性心力衰竭患者应密切监测高钾血症。

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