Department of Cognitive,Linguistic and Psychological Sciences,Brown University,Providence,RI
Behav Brain Sci. 2014 Dec;37(6):561-2; discussion 577-604. doi: 10.1017/S0140525X13004093.
Ackermann et al. disregard attested knowledge concerning aphasia, Parkinson disease, cortical-to-striatal circuits, basal ganglia, laryngeal phonation, and other matters. Their dual-pathway model cannot account for "what is special about the human brain." Their human cortical-to-laryngeal neural circuit does not exist. Basal ganglia operations, enhanced by mutations on FOXP2, confer human motor-control, linguistic, and cognitive capabilities.
阿克曼等人忽视了有关失语症、帕金森病、皮质-纹状体回路、基底神经节、喉发声以及其他事项的确切知识。他们的双通路模型无法解释“人类大脑的独特之处”。他们的人类皮质-喉神经回路并不存在。FOXP2 基因突变增强了基底神经节的运作，赋予了人类运动控制、语言和认知能力。
