Reitano R, Valenti-Milito M, Barbagallo F, Torre D, Raimondi F, Siciliano R, Ferrauto A
G Clin Med. 1989 Aug-Sep;70(8-9):497, 499-501.
The high angiotensin-converting activity, in 13 of the 17 patients afflicted with hepatic cirrhosis, has been put in relation to the hypoxia influence. The insufficient cession of O2 seems to stimulate the enzyme activity, especially for the patients who simultaneously present low values of blood pseudo-cholinesterase. The AA think that this behaviour is to be ascribed to a deficiency of the hepatic proteic synthesis of vasoactive substances, such as the angiotensins.
在17例肝硬化患者中，有13例血管紧张素转换活性较高，这与缺氧影响有关。氧气供应不足似乎会刺激酶活性，尤其是对于同时血液假性胆碱酯酶值较低的患者。研究人员认为，这种现象应归因于肝脏中血管活性物质（如血管紧张素）蛋白质合成的缺乏。
