Barabash R D, Normanskiĭ V E, Andreeva K P, Galil-Ogly G A, Kolobanov A S
Biull Eksp Biol Med. 1989 Oct;108(10):435-7.
By means of morphological methods the mechanisms of thanatogenesis were studied in 57 white mice after the hematoporphyrin derivative administration and xenon lamp radiation imitating the sun spectrum. In dependence of integrative doses of sensitized photoradiation and individual resistance of the organism there was observed immediate (hours), retained (days) and remote (weeks) death of animals. In the first two groups death came as a result of photocoagulation of plasma proteins and toxemic shock with liver, lung and brain injury, and in the third group--as a result of suppurative resorptive intoxication against the background of ulcerous skin necrosis.
通过形态学方法，在给57只小白鼠注射血卟啉衍生物并进行模拟太阳光谱的氙灯照射后，研究了死亡发生机制。根据致敏光辐射的累积剂量和机体的个体抵抗力，观察到动物出现即时（数小时）、持续（数天）和远期（数周）死亡。在前两组中，死亡是血浆蛋白光凝以及伴有肝、肺和脑损伤的毒血症休克所致，而在第三组中，死亡是在溃疡性皮肤坏死背景下的化脓性吸收中毒所致。
