Garcia O L, Castellanos A, Sung R J, Gelband H
Am Heart J. 1978 Nov;96(5):662-8. doi: 10.1016/0002-8703(78)90204-1.
In two infants with Wolff-Parkinson-White type B, right bundle branch block was concealed during sinus rhythm and pacing from close to the atrial entrance of the right-sided accessory pathway. However, pacing from the vicinity of the A-V node, the A-V node itself, and the His bundle exposed the right bundle branch block by producing exclusive ventricular activation through the normal, A-V nodal His-Purkinje pathway. In addition, pacing from close to the A-V node also resulted in fusion beats characterized by absence of delta waves with (pseudo) normal QRS complexes and short H-V intervals. False patterns of tachycardia-dependent and bradycardia-dependent block in the accessory pathway also occurred. These dynamic phenomena were attributed to the (peri-A-V nodal) pacing-related, relatively early arrival of excitation at the ventricles through the normal pathways coexisting with delayed arrival of excitation via the accessory pathway. The latter in turn was due to the longer intra-atrial conduction time from paced (peri-A-V nodal) site to atrial entrance of the accessory pathway.
在两名B型预激综合征婴儿中,窦性心律时以及从靠近右侧旁路心房入口处起搏时,右束支传导阻滞被隐匿。然而,从房室结附近、房室结本身以及希氏束起搏时,通过正常的房室结希氏 - 浦肯野通路产生单独的心室激动,从而暴露了右束支传导阻滞。此外,从靠近房室结处起搏还导致了融合波,其特征为无δ波,伴有(假性)正常QRS波群和短H - V间期。旁路中还出现了与心动过速相关和与心动过缓相关的阻滞的假性模式。这些动态现象归因于(房室结周围)起搏相关的、激动通过正常通路相对较早到达心室,同时激动经旁路延迟到达。后者又是由于从起搏(房室结周围)部位到旁路心房入口的心房内传导时间较长所致。