Re: Sodium Valproate-Induced Myopathy in a Child.
作者信息
Finsterer Josef, Frank Marlies
机构信息
Departments of Neurology, Krankenanstalt Rudolfstiftng, Vienna, Austria.
First Medical, Krankenanstalt Rudolfstiftng, Vienna, Austria.
出版信息
Sultan Qaboos Univ Med J. 2015 Aug;15(3):e442-3. doi: 10.18295/squmj.2015.15.03.025. Epub 2015 Aug 24.
Abstract
摘要
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本文引用的文献
1
Sodium valproate - Induced skeletal myopathy.
Indian J Pediatr. 2005 Mar;72(3):243-244. doi: 10.1007/BF02859266.
2
Sodium valproate-induced myopathy in a child.
Sultan Qaboos Univ Med J. 2015 Feb;15(1):e146-7. Epub 2015 Jan 21.
3
Mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes unveiled by valproate.
J Pediatr Neurosci. 2013 May;8(2):135-7. doi: 10.4103/1817-1745.117847.
4
Valproate-induced hyperammonemic encephalopathy followed by benzodiazepine withdrawal in a patient with schizoaffective disorder: a differential diagnosis.
J Neuropsychiatry Clin Neurosci. 2013 Summer;25(3):E69-70. doi: 10.1176/appi.neuropsych.12090218.
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Drug-resistant epilepsia and fulminant valproate liver toxicity. Alpers-Huttenlocher syndrome in two children confirmed post mortem by identification of p.W748S mutation in POLG gene.
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Activities of respiratory chain complexes and citrate synthase influenced by pharmacologically different antidepressants and mood stabilizers.
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Valproic acid metabolites inhibit dihydrolipoyl dehydrogenase activity leading to impaired 2-oxoglutarate-driven oxidative phosphorylation.
Biochim Biophys Acta. 2007 Sep;1767(9):1126-33. doi: 10.1016/j.bbabio.2007.06.007. Epub 2007 Jul 10.
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Valproic acid aggravates epilepsy due to MELAS in a patient with an A3243G mutation of mitochondrial DNA.
Metab Brain Dis. 2007 Mar;22(1):105-9. doi: 10.1007/s11011-006-9039-9. Epub 2007 Jan 17.
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[Evolution until death of two members of a family with A3243G mutation and MELAS phenotype versus diabetes mellitus].
Neurologia. 2006 Jul-Aug;21(6):327-32.
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