早期精神分裂症自我障碍的神经生物学模型
Neurobiological Models of Self-Disorders in Early Schizophrenia.
作者信息
Mishara A, Bonoldi I, Allen P, Rutigliano G, Perez J, Fusar-Poli P, McGuire P
机构信息
Department of Clinical Psychology, Chicago School of Professional Psychology, Southern California Campus, Los Angeles, CA;
Department of Psychosis Studies, Institute of Psychiatry, Psychology, and Neuroscience, King's College of London, London, UK; OASIS service, South London and the Maudsley NHS Foundation Trust, London, UK; Department of Brain and Behavioural Sciences, University of Pavia, Italy;
出版信息
Schizophr Bull. 2016 Jul;42(4):874-80. doi: 10.1093/schbul/sbv123. Epub 2015 Sep 18.
Self-disorders (SDs) (from the German Ichstörungen) are alterations of the first-person perspective, long associated with schizophrenia, particularly in early phases. Although psychopathological features of SDs continue to be studied, their neurobiological underpinnings are unknown. This makes it difficult to integrate SDs into contemporary models of psychosis. The present review aims to address this issue, starting from an historical excursus revealing an interconnection between neuroscientific models and the origin of the psychopathological concept of SDs. Subsequently, the more recent neurobiological models related to SDs are discussed, particularly with respect to the onset of schizophrenia.
自我障碍（Self-disorders，SDs，源自德语Ichstörungen）是第一人称视角的改变，长期以来与精神分裂症相关，尤其是在早期阶段。尽管自我障碍的精神病理学特征仍在研究中，但其神经生物学基础尚不清楚。这使得将自我障碍纳入当代精神病模型变得困难。本综述旨在解决这一问题，首先进行历史回顾，揭示神经科学模型与自我障碍精神病理学概念起源之间的联系。随后，讨论了与自我障碍相关的最新神经生物学模型，特别是关于精神分裂症的发病。
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