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一种具有生物活性的非ATP竞争性p38丝裂原活化蛋白激酶抑制剂的发现与表征

Discovery and Characterization of a Biologically Active Non-ATP-Competitive p38 MAP Kinase Inhibitor.

作者信息

Wilson Brice A P, Alam Muhammad S, Guszczynski Tad, Jakob Michal, Shenoy Shilpa R, Mitchell Carter A, Goncharova Ekaterina I, Evans Jason R, Wipf Peter, Liu Gang, Ashwell Jonathan D, O'Keefe Barry R

机构信息

Molecular Targets Laboratory, Center for Cancer Research, Frederick National Laboratory for Cancer Research, National Cancer Institute, National Institutes of Health, Frederick, MD, USA.

Laboratory of Immune Cell Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA.

出版信息

J Biomol Screen. 2016 Mar;21(3):277-89. doi: 10.1177/1087057115615518. Epub 2015 Nov 4.

Abstract

Mitogen-activated protein kinase (MAPK) p38 is part of a broad and ubiquitously expressed family of MAPKs whose activity is responsible for mediating an intracellular response to extracellular stimuli through a phosphorylation cascade. p38 is central to this signaling node and is activated by upstream kinases while being responsible for activating downstream kinases and transcription factors via phosphorylation. Dysregulated p38 activity is associated with numerous autoimmune disorders and has been implicated in the progression of several types of cancer. A number of p38 inhibitors have been tested in clinical trials, with none receiving regulatory approval. One characteristic shared by all of the compounds that failed clinical trials is that they are all adenosine triphosphate (ATP)-competitive p38 inhibitors. Seeing this lack of mechanistic diversity as an opportunity, we screened ~32,000 substances in search of novel p38 inhibitors. Among the inhibitors discovered is a compound that is both non-ATP competitive and biologically active in cell-based models for p38 activity. This is the first reported discovery of a non-ATP-competitive p38 inhibitor that is active in cells and, as such, may enable new pharmacophore designs for both therapeutic and basic research to better understand and exploit non-ATP-competitive inhibitors of p38 activity.

摘要

丝裂原活化蛋白激酶(MAPK)p38是MAPK家族中的一员,该家族广泛且普遍表达,其活性通过磷酸化级联反应介导细胞对细胞外刺激的胞内应答。p38是此信号节点的核心,由上游激酶激活,同时通过磷酸化负责激活下游激酶和转录因子。p38活性失调与多种自身免疫性疾病相关,并与几种类型癌症的进展有关。许多p38抑制剂已在临床试验中进行测试,但均未获得监管批准。所有未能通过临床试验的化合物的一个共同特征是它们都是三磷酸腺苷(ATP)竞争性p38抑制剂。鉴于这种机制多样性的缺乏是一个机会,我们筛选了约32000种物质以寻找新型p38抑制剂。在发现的抑制剂中有一种化合物,它在基于细胞的p38活性模型中既非ATP竞争性又具有生物活性。这是首次报道在细胞中具有活性的非ATP竞争性p38抑制剂的发现,因此,这可能为治疗和基础研究带来新的药效团设计,以更好地理解和利用p38活性的非ATP竞争性抑制剂。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/dfef/7480010/646896048843/nihms-1619132-f0001.jpg

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