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在朱宁病毒对非洲绿猴肾细胞和人肺癌细胞的急性感染过程中不存在超感染排斥现象。

Superinfection exclusion is absent during acute Junin virus infection of Vero and A549 cells.

作者信息

Gaudin Raphaël, Kirchhausen Tomas

机构信息

Department of Cell Biology, Harvard Medical School and Program in Cellular and Molecular Medicine, Boston Children's Hospital, 200 Longwood Ave, Boston, MA 02115, USA.

Department of Pediatrics, Harvard Medical School, 200 Longwood Ave, Boston, MA 02115, USA.

出版信息

Sci Rep. 2015 Nov 9;5:15990. doi: 10.1038/srep15990.

DOI:10.1038/srep15990
PMID:26549784
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4637830/
Abstract

Many viruses have evolved strategies of so-called "superinfection exclusion" to prevent re-infection of a cell that the same virus has already infected. Although Old World arenavirus infection results in down-regulation of its viral receptor and thus superinfection exclusion, whether New World arenaviruses have evolved such a mechanism remains unclear. Here we show that acute infection by the New World Junin virus (JUNV) failed to down-regulate the transferrin receptor and did not induce superinfection exclusion. We observed that Vero cells infected by a first round of JUNV (Candid1 strain) preserve an ability to internalize new incoming JUNV particles that is comparable to that of non-infected cells. Moreover, we developed a dual infection assay with the wild-type Candid1 JUNV and a recombinant JUNV-GFP virus to discriminate between first and second infections at the transcriptional and translational levels. We found that Vero and A549 cells already infected by JUNV were fully competent to transcribe viral RNA from a second round of infection. Furthermore, flow cytometry analysis of viral protein expression indicated that viral translation was normal, regardless of whether cells were previously infected or not. We conclude that in acutely infected cells, Junin virus lacks a superinfection exclusion mechanism.

摘要

许多病毒已经进化出所谓的“超感染排除”策略,以防止同一病毒已经感染的细胞再次被感染。尽管旧大陆沙粒病毒感染会导致其病毒受体下调,从而实现超感染排除,但新大陆沙粒病毒是否进化出了这样一种机制仍不清楚。在这里,我们表明,新大陆胡宁病毒(JUNV)的急性感染未能下调转铁蛋白受体,也未诱导超感染排除。我们观察到,第一轮感染JUNV(Candid1株)的Vero细胞保留了内化新进入的JUNV颗粒的能力,这与未感染细胞的能力相当。此外,我们开发了一种用野生型Candid1 JUNV和重组JUNV-GFP病毒进行的双重感染试验,以在转录和翻译水平上区分第一次和第二次感染。我们发现,已经被JUNV感染的Vero和A549细胞完全有能力转录第二轮感染的病毒RNA。此外,对病毒蛋白表达的流式细胞术分析表明,无论细胞之前是否被感染,病毒翻译都是正常的。我们得出结论,在急性感染的细胞中,胡宁病毒缺乏超感染排除机制。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/c175baa41252/srep15990-f4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/612c9e202e31/srep15990-f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/b333e6aff86a/srep15990-f2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/678451b265c7/srep15990-f3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/c175baa41252/srep15990-f4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/612c9e202e31/srep15990-f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/b333e6aff86a/srep15990-f2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/678451b265c7/srep15990-f3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/ea60/4637830/c175baa41252/srep15990-f4.jpg

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Sorting of small infectious virus particles by flow virometry reveals distinct infectivity profiles.通过流式病毒分析对小型传染性病毒颗粒进行分选,揭示了不同的感染性特征。
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Xapuri virus, a novel mammarenavirus: natural reassortment and increased diversity between New World viruses.雅布瑞拉病毒,一种新型哺乳动物沙粒病毒:新世界病毒之间的自然重配和多样性增加。
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