缓激肽在肠道源性内毒素休克中的作用。
The role of bradykinin in endotoxic shock of intestinal origin.
作者信息
Fink R L, Mashford M L, Caridis D T
出版信息
Aust N Z J Surg. 1977 Feb;47(1):102-5. doi: 10.1111/j.1445-2197.1977.tb03946.x.
Abstract
A relationship was looked for between blood levels of bradykinin and endotoxaemia produced by superior mesenteric artery occlusion in the rabbit. The levels of bradykinin were unrelated to and unaltered by the degree of endotoxaemia observed in the systemic and portal circulations. These findings suggest that bradykinin is not responsible for the release of endotoxin from the gut, or the circulatory collapse which follows.
摘要
研究了兔肠系膜上动脉闭塞所致内毒素血症与缓激肽血浓度之间的关系。观察到，全身循环和门静脉循环中的缓激肽浓度与内毒素血症程度无关，且未受其影响。这些发现表明，缓激肽既不引起肠道内毒素的释放，也不导致随后发生的循环衰竭。
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