Weinberger L M
West J Med. 1977 Aug;127(2):99-103.
Traumatic fibromyositis is not an inflammation; there is no fever, leukocytosis or increased sedimentation rate; electrical characteristics and serum enzyme levels are within normal limits, and there are no observable pathologic alterations, although they have been carefully searched for. Recent attempts to express the effects of muscular sprain or strain as a biochemical disturbance expressed in an unusual pattern of lactate dehydrogenase isoenzymes appear not only to be technically flawed but inconsistent with results of conventional enzyme studies on other muscle and interstitial inflammations. In the author's view, "traumatic" fibromyositis is no more than a verbal construct arrived at by adding an adjectival modifier to the old terms for idiopathic rheumatic disorders. An examination of the evolution of the concept of traumatic fibromyositis shows that it lacks validity as a clinical diagnosis and ought to be abandoned.
创伤性纤维肌炎并非炎症;不存在发热、白细胞增多或血沉加快的情况;电生理特征和血清酶水平均在正常范围内,且尽管已进行仔细检查,但未发现可观察到的病理改变。近期试图将肌肉扭伤或拉伤的影响表述为乳酸脱氢酶同工酶异常模式所体现的生化紊乱,这不仅在技术上存在缺陷,而且与其他肌肉和间质性炎症的传统酶学研究结果不一致。在作者看来,“创伤性”纤维肌炎不过是在特发性风湿性疾病的旧有术语前添加一个形容词修饰词而形成的一种文字构建。对创伤性纤维肌炎概念演变的审视表明,它作为一种临床诊断缺乏有效性,应当被摒弃。