Gutschow Susan E, Walker Christopher M, Martínez-Jiménez Santiago, Rosado-de-Christenson Melissa L, Stowell Justin, Kunin Jeffrey R
From the Department of Radiology, University of Missouri-Kansas City, Kansas City, Mo (S.E.G., C.M.W., S.M.J., M.L.R.d.C., J.S., J.R.K.); and Department of Radiology, Thoracic Imaging Division, Saint Luke's Hospital, 4401 Wornall Rd, Kansas City, MO 64111 (S.E.G., C.M.W., S.M.J., M.L.R.d.C., J.R.K.).
Radiographics. 2016 May-Jun;36(3):660-74. doi: 10.1148/rg.2016150094.
Intramural hematoma (IMH) is included in the spectrum of acute aortic syndrome and appears as an area of hyperattenuating crescentic thickening in the aortic wall that is best seen at nonenhanced computed tomography. IMH is historically believed to originate from ruptured vasa vasorum in the aortic media without an intimal tear, but there are reports of small intimomedial tears identified prospectively at imaging or found at surgery in some cases of IMH. These reports have blurred the distinction between aortic dissection and IMH and raise questions about what truly distinguishes the entities that compose acute aortic syndrome. The pathophysiology of these subgroups and the controversies surrounding their differentiation are discussed. The natural history of IMH is highly variable; it may resolve or progress to aneurysm, dissection, or rupture. The authors review various imaging prognostic factors that should be reported by the radiologist, including Stanford classification, maximum aortic diameter, maximum IMH thickness, focal contrast enhancement (including ulcerlike projection and intramural blood pool), and pleural or pericardial effusion. Medical (nonsurgical) versus surgical treatment strategies depend primarily on the Stanford classification, although more recent studies of Asian cohorts report success of initial medical treatment in patients with Stanford type A IMH, with timed (delayed) surgery for patients who develop complications. Understanding the imaging appearance and prognostic factors of IMH helps the radiologist and surgeon identify patients at greatest risk for complications to ensure appropriate treatment and improve patient outcomes. (©)RSNA, 2016.
壁内血肿(IMH)属于急性主动脉综合征范畴,表现为主动脉壁新月形增厚的高密度区域,在非增强计算机断层扫描中显示最佳。传统上认为IMH起源于主动脉中膜的滋养血管破裂且无内膜撕裂,但有报道称在一些IMH病例中,影像学前瞻性发现或手术中发现了小的内膜中层撕裂。这些报道模糊了主动脉夹层与IMH之间的区别,并引发了关于真正区分构成急性主动脉综合征的实体的问题。本文讨论了这些亚组的病理生理学及其鉴别方面的争议。IMH的自然病程差异很大;它可能消退或进展为动脉瘤、夹层或破裂。作者回顾了放射科医生应报告的各种影像学预后因素,包括斯坦福分类、主动脉最大直径、IMH最大厚度、局灶性对比增强(包括溃疡样突出和壁内血池)以及胸腔或心包积液。内科(非手术)与手术治疗策略主要取决于斯坦福分类,尽管最近对亚洲队列的研究报告称,斯坦福A型IMH患者初始内科治疗取得成功,对出现并发症的患者进行择期(延迟)手术。了解IMH的影像学表现和预后因素有助于放射科医生和外科医生识别并发症风险最高的患者,以确保适当治疗并改善患者预后。(©)RSNA,2016年