Wong Humberto, Desser Terry S, Jeffrey R Brooke
Radiol Case Rep. 2015 Nov 6;3(1):113. doi: 10.2484/rcr.v3i1.113. eCollection 2008.
To describe the appearance of transient hepatic attenuation differences (THADs) of extrahepatic origin.
Five cases of THADs produced by compression of the extrahepatic portal vein at its confluence with the splenic vein were identified prospectively over a four-month period. Two additional cases of peripheral THADs resulting from main portal vein thrombosis were identified from retrospective review of a departmental database.
Streamlining of portal venous flow resulted primarily in left lobar THADs when the portal vein is compressed at its confluence with the splenic vein. THADs were seen in the periphery of the liver in the two cases of main portal vein thrombosis.
Lobar and/or peripheral THADs can be produced by compromise of splenic and extrahepatic portal venous flow. Radiologists should be familiar with the "central pseudotumor" created by the peripheral THAD that can result from portal vein thrombosis.
描述肝外源性短暂性肝实质密度差异(THADs)的表现。
前瞻性地在四个月期间内识别出5例因肝外门静脉在其与脾静脉汇合处受压而产生的THADs。通过回顾部门数据库,从回顾性研究中又识别出2例因门静脉主干血栓形成导致的外周THADs。
当门静脉在其与脾静脉汇合处受压时,门静脉血流的流线化主要导致左叶THADs。在2例门静脉主干血栓形成的病例中,在肝脏周边可见THADs。
脾静脉和肝外门静脉血流受阻可产生叶性和/或外周性THADs。放射科医生应熟悉门静脉血栓形成可能导致的外周THADs所产生的“中央假瘤”。