Franco-Saenz R, Wolffing B K, Rivers R J
Department of Medicine, Medical College of Ohio, Toledo 43699.
Am J Med Sci. 1989 Jun;297(6):385-6. doi: 10.1097/00000441-198906000-00011.
The authors describe a case of hypodipsia and severe hypernatremia most probably secondary to hydrocephalus in a 22-year-old man in the absence of abnormalities of ADH secretion or metabolism. The patient became hypernatremic only in situations when the decreased spontaneous fluid intake was insufficient to replace that lost caused by sweating or vomiting. Adequate hydration returned the sodium value to normal.
作者描述了一例22岁男性患者,患有低渴感和严重高钠血症,最可能继发于脑积水,且不存在抗利尿激素(ADH)分泌或代谢异常。仅在自发液体摄入量减少不足以补充因出汗或呕吐而丢失的液体时,患者才会出现高钠血症。充分补液可使钠值恢复正常。