• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

更正:骨形态发生蛋白4信号传导能够通过诱导SNAIL2在巴雷特食管和食管腺癌中诱导上皮-间质转化样表型。

Correction: BMP4 Signaling Is Able to Induce an Epithelial-Mesenchymal Transition-Like Phenotype in Barrett's Esophagus and Esophageal Adenocarcinoma through Induction of SNAIL2.

作者信息

Kestens Christine, Siersema Peter D, Offerhaus G Johan A, van Baal Jantine W P M

出版信息

PLoS One. 2016 Jun 29;11(6):e0158755. doi: 10.1371/journal.pone.0158755. eCollection 2016.

DOI:10.1371/journal.pone.0158755
PMID:27355344
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4927055/
Abstract

[This corrects the article DOI: 10.1371/journal.pone.0155754.].

摘要

[本文更正了文章的数字对象标识符:10.1371/journal.pone.0155754。]

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b515/4927055/4d306d4ecdba/pone.0158755.g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b515/4927055/8f4d15913e81/pone.0158755.g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b515/4927055/4d306d4ecdba/pone.0158755.g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b515/4927055/8f4d15913e81/pone.0158755.g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b515/4927055/4d306d4ecdba/pone.0158755.g002.jpg

相似文献

1
Correction: BMP4 Signaling Is Able to Induce an Epithelial-Mesenchymal Transition-Like Phenotype in Barrett's Esophagus and Esophageal Adenocarcinoma through Induction of SNAIL2.更正:骨形态发生蛋白4信号传导能够通过诱导SNAIL2在巴雷特食管和食管腺癌中诱导上皮-间质转化样表型。
PLoS One. 2016 Jun 29;11(6):e0158755. doi: 10.1371/journal.pone.0158755. eCollection 2016.
2
Expression of the putative stem cell marker Musashi-1 in Barrett's esophagus and esophageal adenocarcinoma.Musashi-1 作为一种假定的干细胞标志物在 Barrett 食管和食管腺癌中的表达。
Dis Esophagus. 2010 Sep;23(7):580-9. doi: 10.1111/j.1442-2050.2010.01061.x. Epub 2010 Apr 29.
3
Barrett's esophagus and risk of esophageal adenocarcinoma.巴雷特食管与食管腺癌风险
Semin Gastrointest Dis. 2003 Jul;14(3):128-35.
4
Hiatal hernia size, Barrett's length, and severity of acid reflux are all risk factors for esophageal adenocarcinoma.食管裂孔疝大小、巴雷特食管长度以及胃酸反流的严重程度均为食管腺癌的危险因素。
Am J Gastroenterol. 2002 Aug;97(8):1930-6. doi: 10.1111/j.1572-0241.2002.05902.x.
5
Aberrant methylation of secreted frizzled-related protein genes in esophageal adenocarcinoma and Barrett's esophagus.食管腺癌和巴雷特食管中分泌型卷曲相关蛋白基因的异常甲基化。
Int J Cancer. 2005 Sep 10;116(4):584-91. doi: 10.1002/ijc.21045.
6
No association between hOGG1, XRCC1, and XPD polymorphisms and risk of reflux esophagitis, Barrett's esophagus, or esophageal adenocarcinoma: results from the factors influencing the Barrett's adenocarcinoma relationship case-control study.人8-羟基鸟嘌呤DNA糖苷酶1(hOGG1)、X射线修复交叉互补蛋白1(XRCC1)和XPD基因多态性与反流性食管炎、巴雷特食管或食管腺癌风险之间无关联:影响巴雷特腺癌关系病例对照研究的结果
Cancer Epidemiol Biomarkers Prev. 2008 Mar;17(3):736-9. doi: 10.1158/1055-9965.EPI-07-2832.
7
Differential response to preoperative chemoradiation and surgery in esophageal adenocarcinomas based on presence of Barrett's esophagus and symptomatic gastroesophageal reflux.基于巴雷特食管的存在及有症状的胃食管反流情况,食管腺癌对术前放化疗和手术的不同反应
Ann Thorac Surg. 2005 May;79(5):1716-23. doi: 10.1016/j.athoracsur.2004.10.026.
8
Fatty acid synthase expression in Barrett's esophagus: implications for carcinogenesis.巴雷特食管中脂肪酸合酶的表达:对癌变的影响。
J Clin Gastroenterol. 2011 Sep;45(8):665-72. doi: 10.1097/MCG.0b013e318207f240.
9
The value of medical imaging in uncomplicated and complicated Barrett's esophagus.医学成像在单纯性和复杂性巴雷特食管中的价值。
Acta Gastroenterol Belg. 2000 Jan-Mar;63(1):22-8.
10
Expression of the bile acid receptor FXR in Barrett's esophagus and enhancement of apoptosis by guggulsterone in vitro.胆汁酸受体FXR在巴雷特食管中的表达及体外古古甾酮对细胞凋亡的增强作用。
Mol Cancer. 2006 Oct 20;5:48. doi: 10.1186/1476-4598-5-48.

引用本文的文献

1
Abnormal Expression and Prognostic Significance of Bone Morphogenetic Proteins and Their Receptors in Lung Adenocarcinoma.骨形成蛋白及其受体在肺腺癌中的异常表达及预后意义。
Biomed Res Int. 2021 May 7;2021:6663990. doi: 10.1155/2021/6663990. eCollection 2021.

本文引用的文献

1
BMP4 Signaling Is Able to Induce an Epithelial-Mesenchymal Transition-Like Phenotype in Barrett's Esophagus and Esophageal Adenocarcinoma through Induction of SNAIL2.骨形态发生蛋白4信号通路能够通过诱导SNAIL2,在巴雷特食管和食管腺癌中诱导出类似上皮-间质转化的表型。
PLoS One. 2016 May 18;11(5):e0155754. doi: 10.1371/journal.pone.0155754. eCollection 2016.