Braun M, Siauw C, Schirrmeister J, Wirbelauer J
Kinderklinik und Poliklinik, Universitatsklinikum Würzburg, Würzburg.
Z Geburtshilfe Neonatol. 2016 Dec;220(6):265-268. doi: 10.1055/s-0042-109405. Epub 2016 Oct 10.
We report a male newborn who became symptomatic with supraventricular tachycardia on the first day of life. Neither adenosine nor electric cardioversion could terminate the tachycardia, therefore intravenous esmolol (β-receptor blocker) was initiated. Inspite of subsequent administration of various antiarrhythmic medications in increasingly higher doses, repeated supraventricular tachycardic episodes occurred. The electrocardiogram showed typical findings of a multifocal atrial tachycardia as the underlying cause. When tachycardic episodes occurred, they also presented as atrial flutter at 460 bpm and a 2:1 block. Finally, high dosage of amiodarone (10 mg/kgbw/d) led to continuous control of the heart rate without tachycardic episodes. To date our patient is mostly in sinus rhythm but without tachycardic episodes and doing well.
我们报告了一名男性新生儿，其在出生第一天就出现了室上性心动过速症状。腺苷和电复律均无法终止心动过速，因此开始静脉注射艾司洛尔（β受体阻滞剂）。尽管随后以越来越高的剂量使用了各种抗心律失常药物，但仍反复发生室上性心动过速发作。心电图显示潜在病因是多灶性房性心动过速的典型表现。当心动过速发作时，还表现为460次/分的心房扑动和2:1传导阻滞。最后，高剂量的胺碘酮（10mg/kgbw/天）使心率得到持续控制，未再出现心动过速发作。迄今为止，我们的患者大多处于窦性心律，无心动过速发作，情况良好。
