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The Popeye Domain Containing Genes and their Function in Striated Muscle.
J Cardiovasc Dev Dis. 2016 Jun 15;3(2):E22. doi: 10.3390/jcdd3020022.
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Misexpression of Tbx18 in cardiac chambers of fetal mice interferes with chamber-specific developmental programs but does not induce a pacemaker-like gene signature.
J Mol Cell Cardiol. 2016 Aug;97:140-9. doi: 10.1016/j.yjmcc.2016.05.004. Epub 2016 May 11.
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Biology of the Sinus Node and its Disease.
Arrhythm Electrophysiol Rev. 2015 May;4(1):28-34. doi: 10.15420/aer.2015.4.1.28. Epub 2015 May 30.
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Genetic Regulation of Sinoatrial Node Development and Pacemaker Program in the Venous Pole.
J Cardiovasc Dev Dis. 2015 Dec;2(4):282-298. doi: 10.3390/jcdd2040282. Epub 2015 Nov 30.
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POPDC1(S201F) causes muscular dystrophy and arrhythmia by affecting protein trafficking.
J Clin Invest. 2016 Jan;126(1):239-53. doi: 10.1172/JCI79562. Epub 2015 Dec 7.
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Transcription factor ISL1 is essential for pacemaker development and function.
J Clin Invest. 2015 Aug 3;125(8):3256-68. doi: 10.1172/JCI68257. Epub 2015 Jul 20.
7
A common Shox2-Nkx2-5 antagonistic mechanism primes the pacemaker cell fate in the pulmonary vein myocardium and sinoatrial node.
Development. 2015 Jul 15;142(14):2521-32. doi: 10.1242/dev.120220. Epub 2015 Jul 2.
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PLoS One. 2015 Mar 24;10(3):e0118623. doi: 10.1371/journal.pone.0118623. eCollection 2015.
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Fibrosis: a structural modulator of sinoatrial node physiology and dysfunction.
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