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代谢激素在治疗与年龄相关的认知衰退和阿尔茨海默病方面的治疗潜力。

The therapeutic potential of metabolic hormones in the treatment of age-related cognitive decline and Alzheimer's disease.

作者信息

Grizzanti John, Lee Hyoung-Gon, Camins Antoni, Pallas Merce, Casadesus Gemma

机构信息

School of Biomedical Sciences, Kent State University, Kent, OH, USA.

Department of Biology, University of Texas, San Antonio, TX, USA.

出版信息

Nutr Res. 2016 Dec;36(12):1305-1315. doi: 10.1016/j.nutres.2016.11.002. Epub 2016 Nov 8.

Abstract

Aging leads to a number of physiological alterations, specifically changes in circulating hormone levels, increases in fat deposition, decreases in metabolism, changes in inflammatory responses, and reductions in growth factors. These progressive changes in physiology and metabolism are exacerbated by modern culture and Western diet and give rise to diseases such as obesity, metabolic syndrome, and type 2 (non-insulin-dependent) diabetes (T2D). These age and lifestyle-related metabolic diseases are often accompanied by insulin and leptin resistance, as well as aberrant amylin production and signaling. Many of these alterations in hormone production and signaling are directly influenced by an increase in both oxidative stress and inflammation. Importantly, changes in hormone production and signaling have direct effects on brain function and the development of age-related neurologic disorders. Therefore, this review aims to present evidence on the effects that diet and metabolic disease have on age-related cognitive decline and the development of cognitive diseases, particularly Alzheimer disease. This review will focus on the metabolic hormones insulin, leptin, and amylin and their role in cognitive decline, as well as the therapeutic potential of these hormones in treating cognitive disease. Future investigations targeting the long-term effects of insulin and leptin treatment may reveal evidence to reduce risk of cognitive decline and Alzheimer disease.

摘要

衰老会导致一系列生理变化,特别是循环激素水平的改变、脂肪沉积增加、新陈代谢减缓、炎症反应变化以及生长因子减少。现代文化和西方饮食加剧了这些生理和新陈代谢的渐进性变化,进而引发肥胖、代谢综合征和2型(非胰岛素依赖型)糖尿病(T2D)等疾病。这些与年龄和生活方式相关的代谢性疾病通常伴有胰岛素和瘦素抵抗,以及异常的胰岛淀粉样多肽生成和信号传导。激素生成和信号传导的许多这些改变直接受到氧化应激和炎症增加的影响。重要的是,激素生成和信号传导的变化对脑功能以及与年龄相关的神经疾病的发展有直接影响。因此,本综述旨在提供证据,说明饮食和代谢性疾病对与年龄相关的认知衰退和认知疾病,特别是阿尔茨海默病发展的影响。本综述将聚焦于代谢激素胰岛素、瘦素和胰岛淀粉样多肽及其在认知衰退中的作用,以及这些激素在治疗认知疾病方面的治疗潜力。针对胰岛素和瘦素治疗长期效果的未来研究可能会揭示降低认知衰退和阿尔茨海默病风险的证据。

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